Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText.
Kick-your-searchterm to multiple Engines kick-your-query now !>
A dictionary by aggregated review articles of nephrology, medicine and the life sciences
Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

suck abstract from ncbi


10.1111/jcmm.15838

http://scihub22266oqcxt.onion/10.1111/jcmm.15838
suck pdf from google scholar
32949179!7646643!32949179
unlimited free pdf from europmc32949179    free
PDF from PMC    free
html from PMC    free

suck abstract from ncbi


Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534

Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534

Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534

Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534

Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534

Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534
pmid32949179      J+Cell+Mol+Med 2020 ; 24 (21): 12864-12868
Nephropedia Template TP

gab.com Text

Twit Text FOAVip

Twit Text #

English Wikipedia


  • STAT3 isoforms differentially affect ACE2 expression: A potential target for COVID-19 therapy #MMPMID32949179
  • Shamir I; Abutbul-Amitai M; Abbas-Egbariya H; Pasmanik-Chor M; Paret G; Nevo-Caspi Y
  • J Cell Mol Med 2020[Nov]; 24 (21): 12864-12868 PMID32949179show ga
  • The SARS-coronavirus 2 is the aetiologic agent COVID-19. ACE2 has been identified as a cell entry receptor for the virus. Therefore, trying to understand how the gene is controlled has become a major goal. We silenced the expression of STAT3alpha and STAT3beta, and found that while silencing STAT3alpha causes an increase in ACE2 expression, silencing STAT3beta causes the opposite effect. Studying the role of STAT3 in ACE2 expression will shed light on the molecular events that contribute to the progression of the disease and that the different roles of STAT3alpha and STAT3beta in that context must be taken in consideration. Our results place STAT3 in line with additional potential therapeutic targets for treating COVID-19 patients.
  • |Angiotensin-Converting Enzyme 2/genetics/*metabolism[MESH]
  • |Binding Sites[MESH]
  • |COVID-19[MESH]
  • |Humans[MESH]
  • |MCF-7 Cells[MESH]
  • |Promoter Regions, Genetic[MESH]
  • |Protein Isoforms/genetics/metabolism[MESH]
  • |SARS-CoV-2/drug effects[MESH]


  • DeepDyve
  • Pubget Overpricing
  • suck abstract from ncbi

    Linkout box