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suck abstract from ncbi


10.1002/iub.2379

http://scihub22266oqcxt.onion/10.1002/iub.2379
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32936531!ä!32936531

suck abstract from ncbi


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pmid32936531      IUBMB+Life 2020 ; 72 (11): 2331-2354
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  • SARS-CoV-2 multifaceted interaction with the human host Part II: Innate immunity response, immunopathology, and epigenetics #MMPMID32936531
  • Beacon TH; Su RC; Lakowski TM; Delcuve GP; Davie JR
  • IUBMB Life 2020[Nov]; 72 (11): 2331-2354 PMID32936531show ga
  • The SARS-CoV-2 makes its way into the cell via the ACE2 receptor and the proteolytic action of TMPRSS2. In response to the SARS-CoV-2 infection, the innate immune response is the first line of defense, triggering multiple signaling pathways to produce interferons, pro-inflammatory cytokines and chemokines, and initiating the adaptive immune response against the virus. Unsurprisingly, the virus has developed strategies to evade detection, which can result in delayed, excessive activation of the innate immune system. The response elicited by the host depends on multiple factors, including health status, age, and sex. An overactive innate immune response can lead to a cytokine storm, inflammation, and vascular disruption, leading to the vast array of symptoms exhibited by COVID-19 patients. What is known about the expression and epigenetic regulation of the ACE2 gene and the various players in the host response are explored in this review.
  • |*Epigenesis, Genetic[MESH]
  • |Angiotensin-Converting Enzyme 2/*genetics/immunology[MESH]
  • |COVID-19/genetics/*immunology/virology[MESH]
  • |Cytokine Release Syndrome/genetics/*immunology/virology[MESH]
  • |Cytokines/genetics/immunology[MESH]
  • |Gene Expression Regulation[MESH]
  • |Host-Pathogen Interactions/genetics/*immunology[MESH]
  • |Humans[MESH]
  • |Immunity, Innate[MESH]
  • |Interferons/genetics/immunology[MESH]
  • |Receptors, Virus/genetics/immunology[MESH]
  • |SARS-CoV-2/immunology/pathogenicity[MESH]
  • |Serine Endopeptidases/*genetics/immunology[MESH]
  • |Signal Transduction[MESH]
  • |Spike Glycoprotein, Coronavirus/*genetics/immunology[MESH]
  • |Virus Internalization[MESH]


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