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10.1016/j.clim.2020.108591 http://scihub22266oqcxt.onion/10.1016/j.clim.2020.108591 32920210!7482598!32920210     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Immunol 2020 ; 220 (ä): 108591 Nephropedia Template TP {{tp|p=32920210|t=2020. On the genetics and immunopathogenesis of COVID-19 |pdf=|usr=}}{{32920210}} gab.com Text On the genetics and immunopathogenesis of COVID-19 JO: Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=32920210 #FOAvip Most severe cases with COVID-19, especially those with pulmonary failure, are not a consequence of viral burden and/or failure of the 'adaptive' immune response to subdue the pathogen by utilizing an adequate 'adaptive' immune defense. Rather it is a consequence of immunopathology, resulting from imbalanced innate immune response, which may not be linked to pathogen burden at all. In fact, it might be described as an autoinflammatory disease. The Kawasaki-like disease seen in children with SARS-CoV-2 exposure might be another example of similar mechanism. Twit Text FOAVip On the genetics and immunopathogenesis of COVID-19 ????Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=32920210 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32920210 #FOAvip English Wikipedia <ref>{{Cite pmid|32920210}}</ref> On the genetics and immunopathogenesis of COVID-19 #MMPMID32920210 Jacob CO Clin Immunol 2020[Nov]; 220 (ä): 108591 PMID32920210show ga Most severe cases with COVID-19, especially those with pulmonary failure, are not a consequence of viral burden and/or failure of the 'adaptive' immune response to subdue the pathogen by utilizing an adequate 'adaptive' immune defense. Rather it is a consequence of immunopathology, resulting from imbalanced innate immune response, which may not be linked to pathogen burden at all. In fact, it might be described as an autoinflammatory disease. The Kawasaki-like disease seen in children with SARS-CoV-2 exposure might be another example of similar mechanism. |Acute Disease[MESH] |Adaptive Immunity[MESH] |Autoimmunity/*genetics[MESH] |Betacoronavirus/immunology/*pathogenicity[MESH] |CD4-Positive T-Lymphocytes/immunology/pathology/virology[MESH] |CD8-Positive T-Lymphocytes/immunology/pathology/virology[MESH] |COVID-19[MESH] |Coronavirus Infections/genetics/*immunology/physiopathology[MESH] |Cytokine Release Syndrome/genetics/*immunology/physiopathology[MESH] |Host-Pathogen Interactions/genetics/*immunology[MESH] |Humans[MESH] |Immunity, Innate[MESH] |Lymphocyte Activation[MESH] |Mucocutaneous Lymph Node Syndrome/genetics/immunology/physiopathology[MESH] |Pandemics[MESH] |Pneumonia, Viral/genetics/*immunology/physiopathology[MESH] |Respiratory Insufficiency/genetics/*immunology/physiopathology[MESH] |SARS-CoV-2[MESH]On the genetics and immunopathogenesis of COVID-19 (epmc).pdf DeepDyve Pubget Overpricing