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10.1084/jem.20200674 http://scihub22266oqcxt.onion/10.1084/jem.20200674 32910820!7481961!32910820     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Exp+Med 2020 ; 217 (10): ä Nephropedia Template TP {{tp|p=32910820|t=2020. Immune asynchrony in COVID-19 pathogenesis and potential immunotherapies |pdf=|usr=}}{{32910820}} gab.com Text Immune asynchrony in COVID-19 pathogenesis and potential immunotherapies JO: J Exp Med http://www.kidney.de/pget.php?&tw=1&pmid=32910820 #FOAvip The outbreak of coronavirus disease 2019 (COVID-19) is an unprecedented global health crisis. Tissue and peripheral blood analysis indicate profound, aberrant myeloid cell activation, cytokine storm, and lymphopenia, with unknown immunopathological mechanisms. Spatiotemporal control of the quality and quantity of the antiviral immune responses involves synchronized cellular and molecular cascades and cross-talk between innate and adaptive immunity. Dysregulated responses in immunity, such as at the stages of immune sensing, alarming, polarization, and resolution, may contribute to disease pathology. Herein, we approach SARS-CoV-2 through an immunomodulatory lens, discussing possible mechanisms of the asynchronized antiviral immune response and proposing potential therapeutic strategies to correct the dysregulation. Twit Text FOAVip Immune asynchrony in COVID-19 pathogenesis and potential immunotherapies ????J Exp Med http://www.kidney.de/pget.php?&tw=1&pmid=32910820 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32910820 #FOAvip English Wikipedia <ref>{{Cite pmid|32910820}}</ref> Immune asynchrony in COVID-19 pathogenesis and potential immunotherapies #MMPMID32910820 Zhou T; Su TT; Mudianto T; Wang J J Exp Med 2020[Oct]; 217 (10): ä PMID32910820show ga The outbreak of coronavirus disease 2019 (COVID-19) is an unprecedented global health crisis. Tissue and peripheral blood analysis indicate profound, aberrant myeloid cell activation, cytokine storm, and lymphopenia, with unknown immunopathological mechanisms. Spatiotemporal control of the quality and quantity of the antiviral immune responses involves synchronized cellular and molecular cascades and cross-talk between innate and adaptive immunity. Dysregulated responses in immunity, such as at the stages of immune sensing, alarming, polarization, and resolution, may contribute to disease pathology. Herein, we approach SARS-CoV-2 through an immunomodulatory lens, discussing possible mechanisms of the asynchronized antiviral immune response and proposing potential therapeutic strategies to correct the dysregulation. |*Immunotherapy[MESH] |Betacoronavirus/*immunology/physiology[MESH] |COVID-19[MESH] |Coronavirus Infections/*immunology/*therapy[MESH] |Humans[MESH] |Immunity[MESH] |Models, Immunological[MESH] |Pandemics[MESH] |Pneumonia, Viral/*immunology/*therapy[MESH] |SARS-CoV-2[MESH]Immune asynchrony in COVID-19 pathogenesis and potential immunotherapi(epmc).pdf DeepDyve Pubget Overpricing