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10.1080/09537104.2020.1817360 http://scihub22266oqcxt.onion/10.1080/09537104.2020.1817360 32896192!?!32896192 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32896192&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Platelets 2021 ; 32 (3): 314-324 Nephropedia Template TP {{tp|p=32896192|t=2021. Immunothrombosis and thromboinflammation in host defense and disease |pdf=|usr=}}{{32896192}} gab.com Text Immunothrombosis and thromboinflammation in host defense and disease JO: Platelets http://www.kidney.de/pget.php?&tw=1&pmid=32896192 #FOAvip Platelets are increasingly being recognized for playing roles beyond thrombosis and hemostasis. Today we know that they mediate inflammation by direct interactions with innate immune cells or secretion of cytokines/chemokines. Here we review their interactions with neutrophils and monocytes/macrophages in infection and sepsis, stroke, myocardial infarction and venous thromboembolism. We discuss new roles for platelet surface receptors like GPVI or GPIb and also look at platelet contributions to the formation of neutrophil extracellular traps (NETs) as well as to deep vein thrombosis during infection, e.g. in COVID-19 patients. Twit Text FOAVip Immunothrombosis and thromboinflammation in host defense and disease ????Platelets http://www.kidney.de/pget.php?&tw=1&pmid=32896192 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32896192 #FOAvip English Wikipedia <ref>{{Cite pmid|32896192}}</ref> Immunothrombosis and thromboinflammation in host defense and disease #MMPMID32896192 Martinod K; Deppermann C Platelets 2021[Apr]; 32 (3): 314-324 PMID32896192show ga Platelets are increasingly being recognized for playing roles beyond thrombosis and hemostasis. Today we know that they mediate inflammation by direct interactions with innate immune cells or secretion of cytokines/chemokines. Here we review their interactions with neutrophils and monocytes/macrophages in infection and sepsis, stroke, myocardial infarction and venous thromboembolism. We discuss new roles for platelet surface receptors like GPVI or GPIb and also look at platelet contributions to the formation of neutrophil extracellular traps (NETs) as well as to deep vein thrombosis during infection, e.g. in COVID-19 patients. |Blood Platelets/*immunology/pathology[MESH] |COVID-19/genetics/*immunology/pathology/virology[MESH] |Cell Communication/genetics/immunology[MESH] |Cytokines/genetics/immunology[MESH] |Extracellular Traps/genetics/immunology[MESH] |Gene Expression Regulation[MESH] |Humans[MESH] |Inflammation[MESH] |Macrophages/immunology/pathology[MESH] |Monocytes/immunology/pathology[MESH] |Myocardial Infarction/genetics/*immunology/pathology[MESH] |Neutrophils/*immunology/pathology[MESH] |Platelet Glycoprotein GPIb-IX Complex/genetics/immunology[MESH] |Platelet Membrane Glycoproteins/genetics/immunology[MESH] |Sepsis/genetics/*immunology/pathology[MESH] |Stroke/genetics/*immunology/pathology[MESH]Immunothrombosis and thromboinflammation in host defense and disease (epmc).pdf DeepDyve Pubget Overpricing