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10.1016/j.it.2020.08.001

http://scihub22266oqcxt.onion/10.1016/j.it.2020.08.001
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32863134!7416769!32863134
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suck abstract from ncbi


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pmid32863134      Trends+Immunol 2020 ; 41 (10): 856-859
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  • Modeling Potential Autophagy Pathways in COVID-19 and Sarcoidosis #MMPMID32863134
  • Calender A; Israel-Biet D; Valeyre D; Pacheco Y
  • Trends Immunol 2020[Oct]; 41 (10): 856-859 PMID32863134show ga
  • Coronavirus disease 2019 (COVID-19) is caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) and mainly affects the lungs. Sarcoidosis is an autoinflammatory disease characterized by the diffusion of granulomas in the lungs and other organs. Here, we discuss how the two diseases might involve some common mechanistic cellular pathways around the regulation of autophagy.
  • |Angiotensin-Converting Enzyme Inhibitors/therapeutic use[MESH]
  • |Autophagy/*drug effects/genetics[MESH]
  • |Azithromycin/therapeutic use[MESH]
  • |Betacoronavirus/growth & development/*pathogenicity[MESH]
  • |COVID-19[MESH]
  • |Chloroquine/therapeutic use[MESH]
  • |Coronavirus Infections/*drug therapy/epidemiology/genetics/virology[MESH]
  • |Host-Pathogen Interactions/drug effects[MESH]
  • |Humans[MESH]
  • |Isoniazid/therapeutic use[MESH]
  • |Lung/drug effects/pathology/virology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*drug therapy/epidemiology/genetics/virology[MESH]
  • |Pulmonary Edema/*drug therapy/epidemiology/genetics/virology[MESH]
  • |Rifampin/therapeutic use[MESH]
  • |SARS-CoV-2[MESH]
  • |Sarcoidosis/*drug therapy/epidemiology/genetics/virology[MESH]
  • |Severe Acute Respiratory Syndrome/*drug therapy/epidemiology/genetics/virology[MESH]


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