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10.1080/21645515.2020.1799669

http://scihub22266oqcxt.onion/10.1080/21645515.2020.1799669
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32841579!7644241!32841579
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suck abstract from ncbi

pmid32841579      Hum+Vaccin+Immunother 2020 ; 16 (10): 2363-2365
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  • Cytokines and COVID-19: friends or foes? #MMPMID32841579
  • Rokni M; Hamblin MR; Rezaei N
  • Hum Vaccin Immunother 2020[Oct]; 16 (10): 2363-2365 PMID32841579show ga
  • The cytokine storm following sepsis has been proven to be an important mechanism for triggering acute respiratory distress syndrome, which is a fatal uncontrolled systemic inflammation characterized by high concentrations of pro-inflammatory cytokines and chemokines, secreted by immune effector cells. The cytokine storm also occurs in the recently emerged novel coronavirus disease (COVID-19). Therefore, cytokines which usually help the immune system to fight infections are potentially harmful in the course of COVID-19 infections. Therefore, avoiding or mitigating the cytokine storm may be a key treatment for severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2).
  • |Antibodies, Monoclonal, Humanized/*therapeutic use[MESH]
  • |Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*pathology[MESH]
  • |Cytokine Receptor gp130/antagonists & inhibitors[MESH]
  • |Cytokine Release Syndrome/*drug therapy/pathology[MESH]
  • |Cytokines/*blood/metabolism[MESH]
  • |Humans[MESH]
  • |Immunosuppressive Agents/*therapeutic use[MESH]
  • |Inflammation/drug therapy/immunology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*pathology[MESH]
  • |SARS-CoV-2[MESH]


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