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10.1002/ccd.29179 http://scihub22266oqcxt.onion/10.1002/ccd.29179 32767631!7436575!32767631     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Catheter+Cardiovasc+Interv 2021 ; 97 (2): 272-277 Nephropedia Template TP {{tp|p=32767631|t=2021. Acute myocardial infarction and large coronary thrombosis in a patient with COVID-19 |pdf=|usr=}}{{32767631}} gab.com Text Acute myocardial infarction and large coronary thrombosis in a patient with COVID-19 JO: Catheter Cardiovasc Interv http://www.kidney.de/pget.php?&tw=1&pmid=32767631 #FOAvip This is a case report of a 60-year-old male, without any cardiovascular risk factor and no cardiac history admitted to hospital with a diagnosis of interstitial pneumonia caused by coronavirus disease 2019 (COVID-19). After 7 days, the blood tests showed a significant rise of inflammatory and procoagulant markers, along with a relevant elevation of high-sensitivity Troponin I. Electrocardiogram and transthoracic echocardiogram (TTE) were consistent with a diagnosis of infero-posterolateral acute myocardial infarction and the patient was transferred to the isolated Cath Lab for primary percutaneous coronary intervention (PCI). The angiography showed an acute massive thrombosis of a dominant right coronary artery without clear evidence of atherosclerosis. Despite the optimal pharmacological therapies and different PCI techniques, the final TIMI flow was 0/1 and after 3 hr the clinical condition evolved in cardiac arrest for pulseless electric activity. Acute coronary syndrome-ST-elevation myocardial infarction is a relevant complication of COVID-19. Due to high levels of proinflammatory mediators, diffuse coronary thrombosis could occur even in patients without cardiac history or comorbidities. This clinical case suggests that coronary thrombosis in COVID-19 patients may be unresponsive to optimal pharmacological (GP IIb-IIIa infusion) and mechanical treatment (PCI). Twit Text FOAVip Acute myocardial infarction and large coronary thrombosis in a patient with COVID-19 ????Catheter Cardiovasc Interv http://www.kidney.de/pget.php?&tw=1&pmid=32767631 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32767631 #FOAvip English Wikipedia <ref>{{Cite pmid|32767631}}</ref> Acute myocardial infarction and large coronary thrombosis in a patient with COVID-19 #MMPMID32767631 Tedeschi D; Rizzi A; Biscaglia S; Tumscitz C Catheter Cardiovasc Interv 2021[Feb]; 97 (2): 272-277 PMID32767631show ga This is a case report of a 60-year-old male, without any cardiovascular risk factor and no cardiac history admitted to hospital with a diagnosis of interstitial pneumonia caused by coronavirus disease 2019 (COVID-19). After 7 days, the blood tests showed a significant rise of inflammatory and procoagulant markers, along with a relevant elevation of high-sensitivity Troponin I. Electrocardiogram and transthoracic echocardiogram (TTE) were consistent with a diagnosis of infero-posterolateral acute myocardial infarction and the patient was transferred to the isolated Cath Lab for primary percutaneous coronary intervention (PCI). The angiography showed an acute massive thrombosis of a dominant right coronary artery without clear evidence of atherosclerosis. Despite the optimal pharmacological therapies and different PCI techniques, the final TIMI flow was 0/1 and after 3 hr the clinical condition evolved in cardiac arrest for pulseless electric activity. Acute coronary syndrome-ST-elevation myocardial infarction is a relevant complication of COVID-19. Due to high levels of proinflammatory mediators, diffuse coronary thrombosis could occur even in patients without cardiac history or comorbidities. This clinical case suggests that coronary thrombosis in COVID-19 patients may be unresponsive to optimal pharmacological (GP IIb-IIIa infusion) and mechanical treatment (PCI). |COVID-19/*complications/diagnosis/therapy[MESH] |Coronary Angiography[MESH] |Coronary Thrombosis/diagnosis/*therapy/*virology[MESH] |Echocardiography[MESH] |Electrocardiography[MESH] |Eptifibatide/therapeutic use[MESH] |Fatal Outcome[MESH] |Humans[MESH] |Male[MESH] |Middle Aged[MESH] |Myocardial Infarction/diagnosis/*therapy/*virology[MESH] |Percutaneous Coronary Intervention[MESH]Acute myocardial infarction and large coronary thrombosis in a patient(epmc).pdf DeepDyve Pubget Overpricing