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10.1016/j.ijid.2020.07.072

http://scihub22266oqcxt.onion/10.1016/j.ijid.2020.07.072
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32763444!7403134!32763444
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suck abstract from ncbi

pmid32763444      Int+J+Infect+Dis 2020 ; 99 (ä): 276-278
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  • Critical illness myopathy after COVID-19 #MMPMID32763444
  • Bagnato S; Boccagni C; Marino G; Prestandrea C; D'Agostino T; Rubino F
  • Int J Infect Dis 2020[Oct]; 99 (ä): 276-278 PMID32763444show ga
  • This paper describes a patient who developed diffuse and symmetrical muscle weakness after a long stay in the intensive care unit (ICU) due to coronavirus disease 2019 (COVID-19). The patient underwent a neurophysiological protocol, including nerve conduction studies, concentric needle electromyography (EMG) of the proximal and distal muscles, and direct muscle stimulation (DMS). Nerve conduction studies showed normal sensory conduction and low-amplitude compound muscle action potentials (CMAPs). EMG revealed signs of myopathy, which were more pronounced in the lower limbs. The post-DMS CMAP was absent in the quadriceps and of reduced amplitude in the tibialis anterior muscle. Based on these clinical and neurophysiological findings, a diagnosis of critical illness myopathy was made according to the current diagnostic criteria. Given the large number of patients with COVID-19 who require long ICU stays, many are very likely to develop ICU-acquired weakness, as did the patient described here. Health systems must plan to provide adequate access to rehabilitative facilities for both pulmonary and motor rehabilitative treatment after COVID-19.
  • |*Betacoronavirus[MESH]
  • |*Critical Illness[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*complications[MESH]
  • |Female[MESH]
  • |Humans[MESH]
  • |Middle Aged[MESH]
  • |Muscular Diseases/*etiology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*complications[MESH]


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