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10.1556/650.2020.31923 http://scihub22266oqcxt.onion/10.1556/650.2020.31923 32750019!ä!32750019 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32750019&cmd=llinks): Failed to open stream: HTTP request failed! 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Az uj koronavirus (SARS-CoV-2) okozta fertozesben szenvedo vese- es majatultetett betegek ellatasanak specialis szempontjai (A COVID-19-pandemia orvosszakmai kerdesei) |pdf=|usr=}}{{32750019}} gab.com Text Az uj koronavirus (SARS-CoV-2) okozta fertozesben szenvedo vese- es majatultetett betegek ellatasanak specialis szempontjai (A COVID-19-pandemia orvosszakmai kerdesei) JO: Orv Hetil http://www.kidney.de/pget.php?&tw=1&pmid=32750019 #FOAvip Due to the COVID-19 pandemic caused by infection with the novel, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), transplant medicine also had to face a new, hitherto unknown challenge. To be prepared for any possibility, we consider it important to summarize the current knowledge regarding COVID-19 of liver and kidney transplant patients. Very early reports from Spanish and French registry recorded fatality rates of 18.6% and 13%, respectively, in renal patients which suggests a moderately worse outcome compared to the general population. In patients with positive PCR test but not showing clinical signs, the reduction of immunosuppression is not advised. In the case of gastrointestinal or respiratory signs with fever, the discontinuation of mycophenolate or mTOR inhibitors is recommended with decrease of the trough levels of calcineurin inhibitors to the lowest effective limit. Stop (kidney transplanted patients) or decrease (liver transplanted patients) immunosuppression and maintain corticosteroids when pulmonal injury develops and consider anti-IL1 and anti-IL6 monoclonal antibody use when hyperinflammatory syndrome is evolving. No proven effective treatment for SARS-CoV-2 exists currently. The use of lopinavir/ritonavir should be avoided because of the severe drug interaction with calcineurin inhibitors. The efficacy and tolerability of hidroxychloroquin remains to be also questionable; enroll patients into clinical trial with remdesivir or favipiravir if available. COVID-19 is characterized by virus-induced endothelial dysfunction, procoagulant state and renin-angiotensin-aldosteron system imbalance. Early thromboprofilaxis combination with low-molecular-weight heparin and low-dose aspirin is strongly recommended with the maintenance of angiotensin-converting enzyme inhibitor (ACEI) or angiotensin-II-receptor blocker (ARB) therapy when they were prescribed earlier. Orv Hetil. 2020; 161(32): 1310-1321. Twit Text FOAVip Az uj koronavirus (SARS-CoV-2) okozta fertozesben szenvedo vese- es majatultetett betegek ellatasanak specialis szempontjai (A COVID-19-pandemia orvosszakmai kerdesei) ????Orv Hetil http://www.kidney.de/pget.php?&tw=1&pmid=32750019 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32750019 #FOAvip English Wikipedia <ref>{{Cite pmid|32750019}}</ref> Az uj koronavirus (SARS-CoV-2) okozta fertozesben szenvedo vese- es majatultetett betegek ellatasanak specialis szempontjai (A COVID-19-pandemia orvosszakmai kerdesei) #MMPMID32750019 Remport A; Gerlei Z; Cseprekal O; Wagner L; Foldes K; Marton A; Patonai A; Torok S; Haboub-Sandil A; Varga M; Doros A; Smudla A; Fazakas J; Kobori L Orv Hetil 2020[Aug]; 161 (32): 1310-1321 PMID32750019show ga Due to the COVID-19 pandemic caused by infection with the novel, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), transplant medicine also had to face a new, hitherto unknown challenge. To be prepared for any possibility, we consider it important to summarize the current knowledge regarding COVID-19 of liver and kidney transplant patients. Very early reports from Spanish and French registry recorded fatality rates of 18.6% and 13%, respectively, in renal patients which suggests a moderately worse outcome compared to the general population. In patients with positive PCR test but not showing clinical signs, the reduction of immunosuppression is not advised. In the case of gastrointestinal or respiratory signs with fever, the discontinuation of mycophenolate or mTOR inhibitors is recommended with decrease of the trough levels of calcineurin inhibitors to the lowest effective limit. Stop (kidney transplanted patients) or decrease (liver transplanted patients) immunosuppression and maintain corticosteroids when pulmonal injury develops and consider anti-IL1 and anti-IL6 monoclonal antibody use when hyperinflammatory syndrome is evolving. No proven effective treatment for SARS-CoV-2 exists currently. The use of lopinavir/ritonavir should be avoided because of the severe drug interaction with calcineurin inhibitors. The efficacy and tolerability of hidroxychloroquin remains to be also questionable; enroll patients into clinical trial with remdesivir or favipiravir if available. COVID-19 is characterized by virus-induced endothelial dysfunction, procoagulant state and renin-angiotensin-aldosteron system imbalance. Early thromboprofilaxis combination with low-molecular-weight heparin and low-dose aspirin is strongly recommended with the maintenance of angiotensin-converting enzyme inhibitor (ACEI) or angiotensin-II-receptor blocker (ARB) therapy when they were prescribed earlier. Orv Hetil. 2020; 161(32): 1310-1321. |*Kidney Transplantation[MESH] |*Liver Transplantation[MESH] |*Transplant Recipients[MESH] |Adrenal Cortex Hormones/therapeutic use[MESH] |Betacoronavirus[MESH] |COVID-19[MESH] |Calcineurin Inhibitors/adverse effects[MESH] |Contraindications, Drug[MESH] |Coronavirus Infections/*complications[MESH] |Drug Combinations[MESH] |Drug Interactions[MESH] |Humans[MESH] |Immunosuppression Therapy[MESH] |Lopinavir/adverse effects[MESH] |Pandemics[MESH] |Pneumonia, Viral/*complications[MESH] |Ritonavir/adverse effects[MESH]Az uj koronavirus (SARS-CoV-2) okozta fertozesben szenvedo vese- es ma(epmc).pdf DeepDyve Pubget Overpricing