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10.3390/ijerph17155573

http://scihub22266oqcxt.onion/10.3390/ijerph17155573
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suck abstract from ncbi


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pmid32748812      Int+J+Environ+Res+Public+Health 2020 ; 17 (15): ä
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  • Evidence-Based Considerations Exploring Relations between SARS-CoV-2 Pandemic and Air Pollution: Involvement of PM2 5-Mediated Up-Regulation of the Viral Receptor ACE-2 #MMPMID32748812
  • Borro M; Di Girolamo P; Gentile G; De Luca O; Preissner R; Marcolongo A; Ferracuti S; Simmaco M
  • Int J Environ Res Public Health 2020[Aug]; 17 (15): ä PMID32748812show ga
  • The COVID-19/SARS-CoV-2 pandemic struck health, social and economic systems worldwide, and represents an open challenge for scientists -coping with the high inter-individual variability of COVID-19, and for policy makers -coping with the responsibility to understand environmental factors affecting its severity across different geographical areas. Air pollution has been warned of as a modifiable factor contributing to differential SARS-CoV-2 spread but the biological mechanisms underlying the phenomenon are still unknown. Air quality and COVID-19 epidemiological data from 110 Italian provinces were studied by correlation analysis, to evaluate the association between particulate matter (PM)(2.5) concentrations and incidence, mortality rate and case fatality risk of COVID-19 in the period 20 February-31 March 2020. Bioinformatic analysis of the DNA sequence encoding the SARS-CoV-2 cell receptor angiotensin-converting enzyme 2 (ACE-2) was performed to identify consensus motifs for transcription factors mediating cellular response to pollutant insult. Positive correlations between PM(2.5) levels and the incidence (r = 0.67, p < 0.0001), the mortality rate (r = 0.65, p < 0.0001) and the case fatality rate (r = 0.7, p < 0.0001) of COVID-19 were found. The bioinformatic analysis of the ACE-2 gene identified nine putative consensus motifs for the aryl hydrocarbon receptor (AHR). Our results confirm the supposed link between air pollution and the rate and outcome of SARS-CoV-2 infection and support the hypothesis that pollution-induced over-expression of ACE-2 on human airways may favor SARS-CoV-2 infectivity.
  • |Air Pollution/*adverse effects[MESH]
  • |Angiotensin-Converting Enzyme 2[MESH]
  • |Base Sequence[MESH]
  • |Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/epidemiology/*virology[MESH]
  • |Humans[MESH]
  • |Italy/epidemiology[MESH]
  • |Pandemics[MESH]
  • |Particulate Matter/*adverse effects[MESH]
  • |Peptidyl-Dipeptidase A/genetics/*metabolism[MESH]
  • |Pneumonia, Viral/epidemiology/*virology[MESH]
  • |Promoter Regions, Genetic[MESH]
  • |Receptors, Virus/genetics/*metabolism[MESH]
  • |SARS-CoV-2[MESH]


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