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10.1007/s40256-020-00431-z

http://scihub22266oqcxt.onion/10.1007/s40256-020-00431-z
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32748336!7398761!32748336
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suck abstract from ncbi

pmid32748336      Am+J+Cardiovasc+Drugs 2020 ; 20 (5): 393-403
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  • Current Overview on Hypercoagulability in COVID-19 #MMPMID32748336
  • Singhania N; Bansal S; Nimmatoori DP; Ejaz AA; McCullough PA; Singhania G
  • Am J Cardiovasc Drugs 2020[Oct]; 20 (5): 393-403 PMID32748336show ga
  • The coronavirus disease 2019 (COVID-19) pandemic, caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), has brought many unique pathologies, such as coagulopathy, prompting a desperate need for effective management. COVID-19-associated coagulopathy (CAC) can cause various thromboembolic complications, especially in critically ill patients. The pathogenesis is likely due to endothelial injury, immobilization, and an increase in circulating prothrombotic factors. Data on treatment are limited, although prophylactic anticoagulation is advised in all hospitalized patients. Herein, we have comprehensively reviewed the current literature available on CAC and highlight the pathogenesis, clinical features, and management of CAC.
  • |*Blood Coagulation Disorders/etiology/physiopathology/prevention & control[MESH]
  • |*Coronavirus Infections/blood/epidemiology/therapy[MESH]
  • |*Pandemics[MESH]
  • |*Pneumonia, Viral/blood/epidemiology/therapy[MESH]
  • |*Thrombophilia/drug therapy/virology[MESH]
  • |Betacoronavirus/physiology[MESH]
  • |COVID-19[MESH]
  • |Chemoprevention/*methods[MESH]
  • |Hematologic Agents/*pharmacology[MESH]
  • |Humans[MESH]


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