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10.1016/j.redox.2020.101655 http://scihub22266oqcxt.onion/10.1016/j.redox.2020.101655 32738789!7381406!32738789     free     free     free       Redox+Biol 2020 ; 36 (ä): 101655 Nephropedia Template TP {{tp|p=32738789|t=2020. Nox2 activation in Covid-19 |pdf=|usr=}}{{32738789}} gab.com Text Nox2 activation in Covid-19 JO: Redox Biol http://www.kidney.de/pget.php?&tw=1&pmid=32738789 #FOAvip Nox2 is responsible for artery dysfunction via production of reactive oxidant species. RNA viruses may activate Nox2, but it is unknown if this occurs in coronavirus 2019(Covid-19). Nox2 activation by soluble Nox2-derived peptide(sNox2-dp) was measured in patients hospitalized for Covid-19 (n = 182) and controls (n = 91). sNox2-dp values were higher in Covid-19 patients versus controls and in severe versus non severe Covid-19. Patients with thrombotic events(n = 35,19%) had higher sNox2-dp than thrombotic event-free ones. A logistic regression analysis showed that sNox2 and coronary heart disease predicted thrombotic events. Oxidative stress by Nox2 activation is associated severe disease and thrombotic events in Covid-19 patients. Twit Text FOAVip Nox2 activation in Covid-19 ????Redox Biol http://www.kidney.de/pget.php?&tw=1&pmid=32738789 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32738789 #FOAvip English Wikipedia <ref>{{Cite pmid|32738789}}</ref> Nox2 activation in Covid-19 #MMPMID32738789 Violi F; Oliva A; Cangemi R; Ceccarelli G; Pignatelli P; Carnevale R; Cammisotto V; Lichtner M; Alessandri F; De Angelis M; Miele MC; D'Ettorre G; Ruberto F; Venditti M; Pugliese F; Mastroianni CM Redox Biol 2020[Sep]; 36 (ä): 101655 PMID32738789show ga Nox2 is responsible for artery dysfunction via production of reactive oxidant species. RNA viruses may activate Nox2, but it is unknown if this occurs in coronavirus 2019(Covid-19). Nox2 activation by soluble Nox2-derived peptide(sNox2-dp) was measured in patients hospitalized for Covid-19 (n = 182) and controls (n = 91). sNox2-dp values were higher in Covid-19 patients versus controls and in severe versus non severe Covid-19. Patients with thrombotic events(n = 35,19%) had higher sNox2-dp than thrombotic event-free ones. A logistic regression analysis showed that sNox2 and coronary heart disease predicted thrombotic events. Oxidative stress by Nox2 activation is associated severe disease and thrombotic events in Covid-19 patients. |Aged[MESH] |Biomarkers/blood[MESH] |COVID-19[MESH] |Coronavirus Infections/blood/complications/*metabolism/pathology[MESH] |Female[MESH] |Humans[MESH] |Male[MESH] |Middle Aged[MESH] |NADPH Oxidase 2/chemistry/*metabolism[MESH] |Oxidative Stress[MESH] |Pandemics[MESH] |Peptide Fragments/blood[MESH] |Pneumonia, Viral/blood/complications/*metabolism/pathology[MESH]Nox2 activation in Covid-19 (epmc).pdf DeepDyve Pubget Overpricing