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10.1016/j.xkme.2019.06.004 http://scihub22266oqcxt.onion/10.1016/j.xkme.2019.06.004 32734203!7380409!32734203     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32734203&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Kidney+Med 2019 ; 1 (4): 221-225 Nephropedia Template TP {{tp|p=32734203|t=2019. Proliferative Glomerulonephritis With Monoclonal IgG3lambda Deposits: A Case Report of a Rare Cause of Monoclonal Gammopathy of Renal Significance |pdf=|usr=}}{{32734203}} gab.com Text Proliferative Glomerulonephritis With Monoclonal IgG3lambda Deposits: A Case Report of a Rare Cause of Monoclonal Gammopathy of Renal Significance JO: Kidney Med http://www.kidney.de/pget.php?&tw=1&pmid=32734203 #FOAvip Proliferative glomerulonephritis with monoclonal immunoglobulin G (IgG) deposits is a rare monoclonal gammopathy of renal significance with dense deposits on electron microscopy similar to polyclonal immune complex-mediated glomerulonephritis. 70% of patients with proliferative glomerulonephritis with monoclonal IgG are negative for a monoclonal (M) spike, and patients with this condition rarely develop an M spike during follow-up. We report a Chinese man in his 50s who presented with nephrotic syndrome and normal glomerular filtration rate. His first kidney biopsy showed masked IgG3 deposition, such that IgG3 staining was apparent only after digestion by enzyme on paraffin tissue, with a membranoproliferative pattern. During follow-up, his glomerular filtration rate worsened and proteinuria increased. 18 months after the first biopsy, the patient developed an M spike; a second kidney biopsy showed proliferative glomerulonephritis with monoclonal IgG deposits with unmasked IgG3lambda deposition. The patient was successfully treated with bortezomib and dexamethasone, followed by lenalidomide and dexamethasone maintenance therapy. Twit Text FOAVip Proliferative Glomerulonephritis With Monoclonal IgG3lambda Deposits: A Case Report of a Rare Cause of Monoclonal Gammopathy of Renal Significance ????Kidney Med http://www.kidney.de/pget.php?&tw=1&pmid=32734203 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32734203 #FOAvip English Wikipedia <ref>{{Cite pmid|32734203}}</ref> Proliferative Glomerulonephritis With Monoclonal IgG3lambda Deposits: A Case Report of a Rare Cause of Monoclonal Gammopathy of Renal Significance #MMPMID32734203 Yu XJ; Wang MJ; Yong ZH; Ma YY; Wang SX; Zhou FD; Zhao MH Kidney Med 2019[Jul]; 1 (4): 221-225 PMID32734203show ga Proliferative glomerulonephritis with monoclonal immunoglobulin G (IgG) deposits is a rare monoclonal gammopathy of renal significance with dense deposits on electron microscopy similar to polyclonal immune complex-mediated glomerulonephritis. 70% of patients with proliferative glomerulonephritis with monoclonal IgG are negative for a monoclonal (M) spike, and patients with this condition rarely develop an M spike during follow-up. We report a Chinese man in his 50s who presented with nephrotic syndrome and normal glomerular filtration rate. His first kidney biopsy showed masked IgG3 deposition, such that IgG3 staining was apparent only after digestion by enzyme on paraffin tissue, with a membranoproliferative pattern. During follow-up, his glomerular filtration rate worsened and proteinuria increased. 18 months after the first biopsy, the patient developed an M spike; a second kidney biopsy showed proliferative glomerulonephritis with monoclonal IgG deposits with unmasked IgG3lambda deposition. The patient was successfully treated with bortezomib and dexamethasone, followed by lenalidomide and dexamethasone maintenance therapy. ?Proliferative Glomerulonephritis With Monoclonal IgG3lambda Deposits: (epmc).pdf DeepDyve Pubget Overpricing