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10.1038/s41577-020-0407-1 http://scihub22266oqcxt.onion/10.1038/s41577-020-0407-1 32728221!7388427!32728221     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Nat+Rev+Immunol 2020 ; 20 (9): 515-516 Nephropedia Template TP {{tp|p=32728221|t=2020. Tissue damage from neutrophil-induced oxidative stress in COVID-19 |pdf=|usr=}}{{32728221}} gab.com Text Tissue damage from neutrophil-induced oxidative stress in COVID-19 JO: Nat Rev Immunol http://www.kidney.de/pget.php?&tw=1&pmid=32728221 #FOAvip The high neutrophil to lymphocyte ratio observed in critically ill patients with COVID-19 is associated with excessive levels of reactive oxygen species (ROS), which promote a cascade of biological events that drive pathological host responses. ROS induce tissue damage, thrombosis and red blood cell dysfunction, which contribute to COVID-19 disease severity. We suggest that free radical scavengers could be beneficial for the most vulnerable patients. Twit Text FOAVip Tissue damage from neutrophil-induced oxidative stress in COVID-19 ????Nat Rev Immunol http://www.kidney.de/pget.php?&tw=1&pmid=32728221 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32728221 #FOAvip English Wikipedia <ref>{{Cite pmid|32728221}}</ref> Tissue damage from neutrophil-induced oxidative stress in COVID-19 #MMPMID32728221 Laforge M; Elbim C; Frere C; Hemadi M; Massaad C; Nuss P; Benoliel JJ; Becker C Nat Rev Immunol 2020[Sep]; 20 (9): 515-516 PMID32728221show ga The high neutrophil to lymphocyte ratio observed in critically ill patients with COVID-19 is associated with excessive levels of reactive oxygen species (ROS), which promote a cascade of biological events that drive pathological host responses. ROS induce tissue damage, thrombosis and red blood cell dysfunction, which contribute to COVID-19 disease severity. We suggest that free radical scavengers could be beneficial for the most vulnerable patients. |Acetylcysteine/therapeutic use[MESH] |Antioxidants/*therapeutic use[MESH] |Betacoronavirus/drug effects/immunology/*pathogenicity[MESH] |COVID-19[MESH] |Coronavirus Infections/*drug therapy/immunology/virology[MESH] |Cytokines/genetics/immunology[MESH] |Drug Therapy, Combination[MESH] |Extracellular Traps/drug effects/immunology/metabolism[MESH] |Gene Expression Regulation[MESH] |Glycine/analogs & derivatives/therapeutic use[MESH] |Host-Pathogen Interactions/drug effects[MESH] |Humans[MESH] |Immunity, Innate/drug effects[MESH] |Lung/drug effects/*immunology/pathology/virology[MESH] |Lymphocytes/drug effects/immunology/virology[MESH] |NF-E2-Related Factor 2/genetics/immunology[MESH] |NF-kappa B/genetics/immunology[MESH] |Neutrophils/*drug effects/immunology/virology[MESH] |Oxidative Stress/drug effects[MESH] |Pandemics[MESH] |Pneumonia, Viral/*drug therapy/immunology/virology[MESH] |Proteinase Inhibitory Proteins, Secretory/therapeutic use[MESH] |Reactive Oxygen Species/antagonists & inhibitors/immunology/metabolism[MESH] |SARS-CoV-2[MESH] |Severity of Illness Index[MESH] |Sulfonamides/therapeutic use[MESH]Tissue damage from neutrophil-induced oxidative stress in COVID-19 (epmc).pdf DeepDyve Pubget Overpricing