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10.1080/1744666X.2020.1803064 http://scihub22266oqcxt.onion/10.1080/1744666X.2020.1803064 32722946!7594185!32722946     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32722946&cmd=llinks): Failed to open stream: HTTP request failed! 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Pathogenesis and treatment of idiopathic and rheumatoid arthritis-related interstitial pneumonia The possible lesson from COVID-19 pneumonia |pdf=|usr=}}{{32722946}} gab.com Text Pathogenesis and treatment of idiopathic and rheumatoid arthritis-related interstitial pneumonia The possible lesson from COVID-19 pneumonia JO: Expert Rev Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=32722946 #FOAvip INTRODUCTION: Main clinical manifestations of SARS-CoV-2 infection are characterized by fever, dyspnea, and interstitial pneumonia, frequently evolving in acute respiratory distress syndrome (ARDS). AREAS COVERED: Features of coronavirus disease 2019 (COVID-19) presents some common points with interstitial lung disease (ILD) both idiopathic and related to rheumatoid arthritis (RA), typically characterized by a chronic progression over time and possibly complicated by acute exacerbation (AE). The study of common pathogenetic mechanisms, such as the involvement of toll-like receptor 4, could contribute to the knowledge and treatment of idiopathic and RA-ILD. Moreover, hyperinflammation, mainly characterized by increase of effector T-cells and inflammatory cytokines, and activation of coagulation cascade, observed in COVID-19 related ARDS have been already shown in patients with AE of idiopathic and RA-ILD. A literature search was performed in PubMed, Embase, Scopus, and Web of Science, together with a manual search in COVID-resource centers of the main journals. EXPERT OPINION: Despite the uncertainty about pathogenetic aspects about COVID-19- pneumonia, it could be a possible model for other forms of ILD and AE. The great amount of data from studies on COVID-19 could be helpful in proposing safe therapeutic approaches for RA-ILD, in understanding pathogenesis of usual interstitial pneumonia and to develop new therapeutic strategies for AE. Twit Text FOAVip Pathogenesis and treatment of idiopathic and rheumatoid arthritis-related interstitial pneumonia The possible lesson from COVID-19 pneumonia ????Expert Rev Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=32722946 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32722946 #FOAvip English Wikipedia <ref>{{Cite pmid|32722946}}</ref> Pathogenesis and treatment of idiopathic and rheumatoid arthritis-related interstitial pneumonia The possible lesson from COVID-19 pneumonia #MMPMID32722946 Manfredi A; Luppi F; Cassone G; Vacchi C; Salvarani C; Sebastiani M Expert Rev Clin Immunol 2020[Aug]; 16 (8): 751-770 PMID32722946show ga INTRODUCTION: Main clinical manifestations of SARS-CoV-2 infection are characterized by fever, dyspnea, and interstitial pneumonia, frequently evolving in acute respiratory distress syndrome (ARDS). AREAS COVERED: Features of coronavirus disease 2019 (COVID-19) presents some common points with interstitial lung disease (ILD) both idiopathic and related to rheumatoid arthritis (RA), typically characterized by a chronic progression over time and possibly complicated by acute exacerbation (AE). The study of common pathogenetic mechanisms, such as the involvement of toll-like receptor 4, could contribute to the knowledge and treatment of idiopathic and RA-ILD. Moreover, hyperinflammation, mainly characterized by increase of effector T-cells and inflammatory cytokines, and activation of coagulation cascade, observed in COVID-19 related ARDS have been already shown in patients with AE of idiopathic and RA-ILD. A literature search was performed in PubMed, Embase, Scopus, and Web of Science, together with a manual search in COVID-resource centers of the main journals. EXPERT OPINION: Despite the uncertainty about pathogenetic aspects about COVID-19- pneumonia, it could be a possible model for other forms of ILD and AE. The great amount of data from studies on COVID-19 could be helpful in proposing safe therapeutic approaches for RA-ILD, in understanding pathogenesis of usual interstitial pneumonia and to develop new therapeutic strategies for AE. |Arthritis, Rheumatoid/*pathology/therapy[MESH] |Betacoronavirus/pathogenicity[MESH] |COVID-19[MESH] |Coronavirus Infections/*pathology/therapy[MESH] |Disease Progression[MESH] |Humans[MESH] |Idiopathic Pulmonary Fibrosis/pathology/therapy[MESH] |Lung Diseases, Interstitial/*pathology/therapy[MESH] |Lung/pathology[MESH] |Pandemics[MESH] |Pneumonia, Viral/*pathology/therapy[MESH] |SARS-CoV-2[MESH] |Symptom Flare Up[MESH]Pathogenesis and treatment of idiopathic and rheumatoid arthritis-rela(epmc).pdf DeepDyve Pubget Overpricing