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10.5802/crbiol.8

http://scihub22266oqcxt.onion/10.5802/crbiol.8
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32720486!ä!32720486

suck abstract from ncbi


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pmid32720486      C+R+Biol 2020 ; 343 (1): 33-39
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  • A nicotinic hypothesis for Covid-19 with preventive and therapeutic implications #MMPMID32720486
  • Changeux JP; Amoura Z; Rey FA; Miyara M
  • C R Biol 2020[Jun]; 343 (1): 33-39 PMID32720486show ga
  • SARS-CoV-2 epidemics raises a considerable issue of public health at the planetary scale. There is a pressing urgency to find treatments based upon currently available scientific knowledge. Therefore, we tentatively propose a hypothesis which hopefully might ultimately help save lives. Based on the current scientific literature and on new epidemiological data which reveal that current smoking status appears to be a protective factor against the infection by SARS-CoV-2 [1], we hypothesize that the nicotinic acetylcholine receptor (nAChR) plays a key role in the pathophysiology of Covid-19 infection and might represent a target for the prevention and control of Covid-19 infection.
  • |*Receptors, Nicotinic[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*physiopathology/prevention & control/*therapy[MESH]
  • |Humans[MESH]
  • |Nicotine/*therapeutic use[MESH]
  • |Nicotinic Agonists/*therapeutic use[MESH]
  • |Pandemics/prevention & control[MESH]
  • |Pneumonia, Viral/*physiopathology/prevention & control/*therapy[MESH]
  • |Smoking[MESH]


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