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suck abstract from ncbi


10.1007/s10067-020-05310-1

http://scihub22266oqcxt.onion/10.1007/s10067-020-05310-1
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32720260!7384868!32720260
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suck abstract from ncbi


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pmid32720260      Clin+Rheumatol 2020 ; 39 (9): 2811-2815
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  • Concomitant new diagnosis of systemic lupus erythematosus and COVID-19 with possible antiphospholipid syndrome Just a coincidence? A case report and review of intertwining pathophysiology #MMPMID32720260
  • Mantovani Cardoso E; Hundal J; Feterman D; Magaldi J
  • Clin Rheumatol 2020[Sep]; 39 (9): 2811-2815 PMID32720260show ga
  • In the midst of the COVID-19 pandemic, further understanding of its complications points towards dysregulated immune response as a major component. Systemic lupus erythematosus (SLE) is also a disease of immune dysregulation leading to multisystem compromise. We present a case of new-onset SLE concomitantly with COVID-19 and development of antiphospholipid antibodies. An 18-year-old female that presented with hemodynamic collapse and respiratory failure, progressed to cardiac arrest, and had a pericardial tamponade drained. She then progressed to severe acute respiratory distress syndrome, severe ventricular dysfunction, and worsening renal function with proteinuria and hematuria. Further studies showed bilateral pleural effusions, positive antinuclear and antidouble-stranded DNA antibodies, lupus anticoagulant, and anticardiolipin B. C3 and C4 levels were low. SARS-Cov-2 PCR was positive after 2 negative tests. She also developed multiple deep venous thrombosis, in the setting of positive antiphospholipid antibodies and lupus anticoagulant. In terms of pathophysiology, COVID-19 is believed to cause a dysregulated cytokine response which could potentially be exacerbated by the shift in Th1 to Th2 response seen in SLE. Also, it is well documented that viral infections are an environmental factor that contributes to the development of autoimmunity; however, COVID-19 is a new entity, and it is not known if it could trigger autoimmune conditions. Additionally, it is possible that SARS-CoV-2, as it happens with other viruses, might lead to the formation of antiphospholipid antibodies, potentially contributing to the increased rates of thrombosis seen in COVID-19.
  • |Adolescent[MESH]
  • |Anemia/etiology[MESH]
  • |Antibodies, Anticardiolipin/immunology[MESH]
  • |Antibodies, Antinuclear/immunology[MESH]
  • |Antibodies, Monoclonal, Humanized/therapeutic use[MESH]
  • |Antiphospholipid Syndrome/complications/diagnosis/*immunology/therapy[MESH]
  • |Anuria/etiology[MESH]
  • |Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Cardiac Tamponade/diagnostic imaging/etiology/therapy[MESH]
  • |Complement C3/immunology[MESH]
  • |Complement C4/immunology[MESH]
  • |Coronavirus Infections/complications/diagnosis/*immunology/therapy[MESH]
  • |DNA/immunology[MESH]
  • |Echocardiography[MESH]
  • |Fatal Outcome[MESH]
  • |Female[MESH]
  • |Heart Arrest/etiology[MESH]
  • |Hematuria/etiology[MESH]
  • |Humans[MESH]
  • |Lupus Coagulation Inhibitor/immunology[MESH]
  • |Lupus Erythematosus, Systemic/blood/complications/diagnosis/*immunology[MESH]
  • |Pandemics[MESH]
  • |Patient Positioning[MESH]
  • |Pericardiocentesis[MESH]
  • |Pneumonia, Viral/complications/diagnosis/*immunology/therapy[MESH]
  • |Prone Position[MESH]
  • |Proteinuria/etiology[MESH]
  • |Renal Dialysis[MESH]
  • |Renal Insufficiency/etiology/therapy[MESH]
  • |Respiration, Artificial[MESH]
  • |Respiratory Distress Syndrome/etiology/therapy[MESH]
  • |Respiratory Insufficiency/etiology/therapy[MESH]
  • |SARS-CoV-2[MESH]
  • |Thrombocytopenia/etiology[MESH]
  • |Venous Thrombosis/etiology[MESH]


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