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10.1038/s41371-020-0387-4 http://scihub22266oqcxt.onion/10.1038/s41371-020-0387-4 32719447!7384729!32719447     free     free     free Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 235.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Hum+Hypertens 2021 ; 35 (1): 4-11 Nephropedia Template TP {{tp|p=32719447|t=2021. Covid-19 and the cardiovascular system: a comprehensive review |pdf=|usr=}}{{32719447}} gab.com Text Covid-19 and the cardiovascular system: a comprehensive review JO: J Hum Hypertens http://www.kidney.de/pget.php?&tw=1&pmid=32719447 #FOAvip Cardiac injury in patients infected with the novel Coronavirus (COVID-19) seems to be associated with higher morbimortality. We provide a broad review of the clinical evolution of COVID-19, emphasizing its impact and implications on the cardiovascular system. The pathophysiology of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is characterized by overproduction of inflammatory cytokines (IL-6 and TNF-alpha) leading to systemic inflammation and multiple organ dysfunction syndrome, acutely affecting the cardiovascular system. Hypertension (56.6%) and diabetes (33.8%) are the most prevalent comorbidities among individuals with COVID-19, who require hospitalization. Furthermore, cardiac injury, defined as elevated us-troponin I, significantly relates to inflammation biomarkers (IL-6 and C-reactive protein (CRP), hyperferritinemia, and leukocytosis), portraying an important correlation between myocardial injury and inflammatory hyperactivity triggered by viral infection. Increased risk for myocardial infarction, fulminant myocarditis rapidly evolving with depressed systolic left ventricle function, arrhythmias, venous thromboembolism, and cardiomyopathies mimicking STEMI presentations are the most prevalent cardiovascular complications described in patients with COVID-19. Moreover, SARS-CoV-2 tropism and interaction with the RAAS system, through ACE2 receptor, possibly enhances inflammation response and cardiac aggression, leading to imperative concerns about the use of ACEi and ARBs in infected patients. Cardiovascular implications result in a worse prognosis in patients with COVID-19, emphasizing the importance of precocious detection and implementation of optimal therapeutic strategies. Twit Text FOAVip Covid-19 and the cardiovascular system: a comprehensive review ????J Hum Hypertens http://www.kidney.de/pget.php?&tw=1&pmid=32719447 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32719447 #FOAvip English Wikipedia <ref>{{Cite pmid|32719447}}</ref> Covid-19 and the cardiovascular system: a comprehensive review #MMPMID32719447 Azevedo RB; Botelho BG; Hollanda JVG; Ferreira LVL; Junqueira de Andrade LZ; Oei SSML; Mello TS; Muxfeldt ES J Hum Hypertens 2021[Jan]; 35 (1): 4-11 PMID32719447show ga Cardiac injury in patients infected with the novel Coronavirus (COVID-19) seems to be associated with higher morbimortality. We provide a broad review of the clinical evolution of COVID-19, emphasizing its impact and implications on the cardiovascular system. The pathophysiology of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is characterized by overproduction of inflammatory cytokines (IL-6 and TNF-alpha) leading to systemic inflammation and multiple organ dysfunction syndrome, acutely affecting the cardiovascular system. Hypertension (56.6%) and diabetes (33.8%) are the most prevalent comorbidities among individuals with COVID-19, who require hospitalization. Furthermore, cardiac injury, defined as elevated us-troponin I, significantly relates to inflammation biomarkers (IL-6 and C-reactive protein (CRP), hyperferritinemia, and leukocytosis), portraying an important correlation between myocardial injury and inflammatory hyperactivity triggered by viral infection. Increased risk for myocardial infarction, fulminant myocarditis rapidly evolving with depressed systolic left ventricle function, arrhythmias, venous thromboembolism, and cardiomyopathies mimicking STEMI presentations are the most prevalent cardiovascular complications described in patients with COVID-19. Moreover, SARS-CoV-2 tropism and interaction with the RAAS system, through ACE2 receptor, possibly enhances inflammation response and cardiac aggression, leading to imperative concerns about the use of ACEi and ARBs in infected patients. Cardiovascular implications result in a worse prognosis in patients with COVID-19, emphasizing the importance of precocious detection and implementation of optimal therapeutic strategies. |Animals[MESH] |COVID-19/epidemiology/physiopathology/*virology[MESH] |Cardiovascular Diseases/epidemiology/physiopathology/*virology[MESH] |Cardiovascular System/physiopathology/*virology[MESH] |Heart Disease Risk Factors[MESH] |Host-Pathogen Interactions[MESH] |Humans[MESH] |Prognosis[MESH] |Renin-Angiotensin System[MESH] |Risk Assessment[MESH]Covid-19 and the cardiovascular system: a comprehensive review (epmc).pdf DeepDyve Pubget Overpricing