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10.1016/j.arteri.2020.06.003 http://scihub22266oqcxt.onion/10.1016/j.arteri.2020.06.003 32718669!7330559!32718669     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Clin+Investig+Arterioscler 2020 ; 32 (6): 278-281 Nephropedia Template TP {{tp|p=32718669|t=2020. Estatinas en COVID-19: inverted question markexiste algun fundamento?|pdf=|usr=}}{{32718669}} gab.com Text Estatinas en COVID-19: inverted question markexiste algun fundamento JO: Clin Investig Arterioscler http://www.kidney.de/pget.php?&tw=1&pmid=32718669 #FOAvip Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the causal agent of coronavirus disease 2019 (COVID-19). Acute respiratory distress syndrome is the main cause of death from COVID-19 and occurs due to an exaggerated inflammatory response that causes the release of pro-inflammatory cytokines such as interleukins and tumor necrosis factor-alpha (TNF-alpha). Statins are lipid lowering drugs with pleiotropic effects. They have shown benefit in the management of inflammatory and autoimmune diseases such as systemic lupus erythematosus, rheumatoid arthritis and multiple sclerosis. Furthermore, due to their immunomodulatory properties, they have been used in the treatment of various infectious diseases such as community-acquired pneumonia and influenza. In this review we analyze the pathophysiological foundations that support the use of statins as an adjunctive treatment in patients with COVID-19. Twit Text FOAVip Estatinas en COVID-19: inverted question markexiste algun fundamento ????Clin Investig Arterioscler http://www.kidney.de/pget.php?&tw=1&pmid=32718669 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32718669 #FOAvip English Wikipedia <ref>{{Cite pmid|32718669}}</ref> Estatinas en COVID-19: inverted question markexiste algun fundamento? #MMPMID32718669 Lima Martinez MM; Contreras MA; Marin W; D'Marco L Clin Investig Arterioscler 2020[Nov]; 32 (6): 278-281 PMID32718669show ga Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the causal agent of coronavirus disease 2019 (COVID-19). Acute respiratory distress syndrome is the main cause of death from COVID-19 and occurs due to an exaggerated inflammatory response that causes the release of pro-inflammatory cytokines such as interleukins and tumor necrosis factor-alpha (TNF-alpha). Statins are lipid lowering drugs with pleiotropic effects. They have shown benefit in the management of inflammatory and autoimmune diseases such as systemic lupus erythematosus, rheumatoid arthritis and multiple sclerosis. Furthermore, due to their immunomodulatory properties, they have been used in the treatment of various infectious diseases such as community-acquired pneumonia and influenza. In this review we analyze the pathophysiological foundations that support the use of statins as an adjunctive treatment in patients with COVID-19. |*COVID-19 Drug Treatment[MESH] |*SARS-CoV-2/physiology[MESH] |Angiotensin-Converting Enzyme 2/metabolism[MESH] |COVID-19/epidemiology[MESH] |Cytokine Release Syndrome/drug therapy[MESH] |Humans[MESH] |Hydroxymethylglutaryl-CoA Reductase Inhibitors/pharmacology/*therapeutic use[MESH] |Liver/drug effects[MESH] |Pandemics[MESH] |Renin-Angiotensin System/physiology[MESH] |Spike Glycoprotein, Coronavirus/metabolism[MESH]Estatinas en COVID-19: inverted question markexiste algun fundamento?(epmc).pdf DeepDyve Pubget Overpricing