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10.1016/j.arcmed.2020.06.012 http://scihub22266oqcxt.onion/10.1016/j.arcmed.2020.06.012 32682575!7303639!32682575     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Arch+Med+Res 2020 ; 51 (7): 608-612 Nephropedia Template TP {{tp|p=32682575|t=2020. Targeting Cytokine Storm to Manage Patients with COVID-19: A Mini-Review |pdf=|usr=}}{{32682575}} gab.com Text Targeting Cytokine Storm to Manage Patients with COVID-19: A Mini-Review JO: Arch Med Res http://www.kidney.de/pget.php?&tw=1&pmid=32682575 #FOAvip Corona Virus Disease 2019 (COVID-19) pandemic is rapidly spreading all over the world. Excessive immune responses trigger life-threatening cytokine release syndrome (CRS) which can result in overproduction of pro-inflammatory cytokines including tumor necrosis factor alpha (TNFalpha), interleukin-6 (IL-6), and IL-1beta with different pro-inflammatory roles. Anecdotal evidence suggests that the modulation of systemic immune responses may have a potential role in the treatment of patients with COVID-19. Given the importance of the issue and the lack of therapeutic treatment or vaccine; anti-cytokine therapy such as IL-6, TNFalpha and IL-1 antagonists have been suggested for the alleviation of hyper-inflammation status in these patients. In this mini-review, we addressed the inflammatory pathways of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) and its relationship with the host cytokine storm. Furthermore, the proposed therapeutic options to reverse hyper-inflammation in infected patients were mentioned. Twit Text FOAVip Targeting Cytokine Storm to Manage Patients with COVID-19: A Mini-Review ????Arch Med Res http://www.kidney.de/pget.php?&tw=1&pmid=32682575 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32682575 #FOAvip English Wikipedia <ref>{{Cite pmid|32682575}}</ref> Targeting Cytokine Storm to Manage Patients with COVID-19: A Mini-Review #MMPMID32682575 Roshanravan N; Seif F; Ostadrahimi A; Pouraghaei M; Ghaffari S Arch Med Res 2020[Oct]; 51 (7): 608-612 PMID32682575show ga Corona Virus Disease 2019 (COVID-19) pandemic is rapidly spreading all over the world. Excessive immune responses trigger life-threatening cytokine release syndrome (CRS) which can result in overproduction of pro-inflammatory cytokines including tumor necrosis factor alpha (TNFalpha), interleukin-6 (IL-6), and IL-1beta with different pro-inflammatory roles. Anecdotal evidence suggests that the modulation of systemic immune responses may have a potential role in the treatment of patients with COVID-19. Given the importance of the issue and the lack of therapeutic treatment or vaccine; anti-cytokine therapy such as IL-6, TNFalpha and IL-1 antagonists have been suggested for the alleviation of hyper-inflammation status in these patients. In this mini-review, we addressed the inflammatory pathways of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) and its relationship with the host cytokine storm. Furthermore, the proposed therapeutic options to reverse hyper-inflammation in infected patients were mentioned. |*COVID-19/complications/immunology/physiopathology/therapy[MESH] |*Cytokine Release Syndrome/immunology/physiopathology/therapy/virology[MESH] |Humans[MESH] |Immunotherapy[MESH]Targeting Cytokine Storm to Manage Patients with COVID-19: A Mini-Revi(epmc).pdf DeepDyve Pubget Overpricing