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10.1016/j.cca.2020.07.019

http://scihub22266oqcxt.onion/10.1016/j.cca.2020.07.019
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32681935!7363601!32681935
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suck abstract from ncbi

pmid32681935      Clin+Chim+Acta 2020 ; 510 (ä): 311-316
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  • SARS-CoV-2 and the cardiovascular system #MMPMID32681935
  • Hafiane A
  • Clin Chim Acta 2020[Nov]; 510 (ä): 311-316 PMID32681935show ga
  • The coronavirus disease COVID-19 is a public health emergency caused by a novel coronavirus named severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2). SARS-CoV-2 infection uses the angiotensin-converting enzyme 2 (ACE2) receptor, and typically spreads through the respiratory tract. Invading viruses can elicit an exaggerated host immune response, frequently leading to a cytokine storm that may be fueling some COVID-19 death. This response contributes to multi-organ dysfunction. Accumulating data points to an increased cardiovascular disease morbidity, and mortality in COVID-19 patients. This brief review explores potential available evidence regarding the association between COVID-19, and cardiovascular complications.
  • |Angiotensin-Converting Enzyme 2[MESH]
  • |Betacoronavirus/*physiology[MESH]
  • |COVID-19[MESH]
  • |Cardiovascular System/drug effects/*virology[MESH]
  • |Coronavirus Infections/complications/drug therapy/metabolism[MESH]
  • |Humans[MESH]
  • |Molecular Targeted Therapy[MESH]
  • |Pandemics[MESH]
  • |Peptidyl-Dipeptidase A/metabolism[MESH]
  • |Pneumonia, Viral/complications/drug therapy/metabolism[MESH]
  • |Renin-Angiotensin System/drug effects[MESH]


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