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10.1161/ATVBAHA.120.314514

http://scihub22266oqcxt.onion/10.1161/ATVBAHA.120.314514
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32657623!7447001!32657623
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suck abstract from ncbi


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pmid32657623      Arterioscler+Thromb+Vasc+Biol 2020 ; 40 (9): 2033-2044
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  • Coagulation Abnormalities and Thrombosis in Patients Infected With SARS-CoV-2 and Other Pandemic Viruses #MMPMID32657623
  • Mackman N; Antoniak S; Wolberg AS; Kasthuri R; Key NS
  • Arterioscler Thromb Vasc Biol 2020[Sep]; 40 (9): 2033-2044 PMID32657623show ga
  • The world is amid a pandemic caused by severe acute respiratory syndrome-coronavirus 2. Severe acute respiratory syndrome-coronavirus causes serious respiratory tract infections that can lead to viral pneumonia, acute respiratory distress syndrome, and death. Some patients with coronavirus disease 2019 (COVID-19) have an activated coagulation system characterized by elevated plasma levels of d-dimer-a biomarker of fibrin degradation. Importantly, high levels of D-dimer on hospital admission are associated with increased risk of mortality. Venous thromboembolism is more common than arterial thromboembolism in hospitalized COVID-19 patients. Pulmonary thrombosis and microvascular thrombosis are observed in autopsy studies, and this may contribute to the severe hypoxia observed in COVID-19 patients. It is likely that multiple systems contribute to thrombosis in COVID-19 patients, such as activation of coagulation, platelet activation, hypofibrinolysis, endothelial cell dysfunction, inflammation, neutrophil extracellular traps, and complement. Targeting these different pathways may reduce thrombosis and improve lung function in COVID-19 patients.
  • |*Betacoronavirus[MESH]
  • |*Blood Coagulation[MESH]
  • |*Pandemics[MESH]
  • |Blood Coagulation Disorders/blood/*complications[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*complications/epidemiology[MESH]
  • |Humans[MESH]
  • |Pneumonia, Viral/*complications/epidemiology[MESH]
  • |SARS-CoV-2[MESH]


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