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10.1161/HYPERTENSIONAHA.120.15464 http://scihub22266oqcxt.onion/10.1161/HYPERTENSIONAHA.120.15464 32654557!ä!32654557 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32654557&cmd=llinks): Failed to open stream: HTTP request failed! 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Association Between Renin-Angiotensin-Aldosterone System Inhibitors and COVID-19 Infection in South Korea |pdf=|usr=}}{{32654557}} gab.com Text Association Between Renin-Angiotensin-Aldosterone System Inhibitors and COVID-19 Infection in South Korea JO: Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=32654557 #FOAvip The severe acute respiratory syndrome coronavirus 2 is known to infect host cells by interacting with ACE2 (angiotensin-converting enzyme 2) expressed in the respiratory epithelium. There have been concerns on whether alterations of ACE2 expression by renin-angiotensin-aldosterone system (RAAS) inhibitors would contribute to the infectivity and severity of coronavirus disease 2019 (COVID-19). We performed a case-control study to investigate the association between RAAS inhibitors and risk and severity of COVID-19 infection in South Korea using the population-based data provided by the Korean National Health Insurance System. Of 16 281 subjects with hypertension, there were 950 (5.8%) confirmed COVID-19 cases. After case-control matching, multivariable-adjusted conditional logistic regression analysis was performed. The adjusted odds ratio and 95% CIs for COVID-19 infection and long-term hospitalization comparing exposure to RAAS inhibitors and nonexposure to RAAS inhibitors was 1.161 (0.958-1.407) and 0.863 (0.533-1.397), respectively. When comparing exposure to RAAS inhibitors and nonexposure to RAAS inhibitors for intensive care unit admission, high-flow oxygen therapy, and death, the adjusted odds ratios (95% CIs) were 1.515 (0.402-5.701), 0.663 (0.272-1.619), and 1.363 (0.513-3.662), respectively. In all analyses, P values were not significant (P>0.05). The present study demonstrates the absence of an identifiable association between the exposure to RAAS inhibitors and risk and severity of COVID-19 infection, supporting the current medical guidelines and recommendations that patients should not discontinue RAAS inhibitors out of a concern that they are at increased risk for infection or severe illness of COVID-19. Twit Text FOAVip Association Between Renin-Angiotensin-Aldosterone System Inhibitors and COVID-19 Infection in South Korea ????Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=32654557 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32654557 #FOAvip English Wikipedia <ref>{{Cite pmid|32654557}}</ref> Association Between Renin-Angiotensin-Aldosterone System Inhibitors and COVID-19 Infection in South Korea #MMPMID32654557 Son M; Seo J; Yang S Hypertension 2020[Sep]; 76 (3): 742-749 PMID32654557show ga The severe acute respiratory syndrome coronavirus 2 is known to infect host cells by interacting with ACE2 (angiotensin-converting enzyme 2) expressed in the respiratory epithelium. There have been concerns on whether alterations of ACE2 expression by renin-angiotensin-aldosterone system (RAAS) inhibitors would contribute to the infectivity and severity of coronavirus disease 2019 (COVID-19). We performed a case-control study to investigate the association between RAAS inhibitors and risk and severity of COVID-19 infection in South Korea using the population-based data provided by the Korean National Health Insurance System. Of 16 281 subjects with hypertension, there were 950 (5.8%) confirmed COVID-19 cases. After case-control matching, multivariable-adjusted conditional logistic regression analysis was performed. The adjusted odds ratio and 95% CIs for COVID-19 infection and long-term hospitalization comparing exposure to RAAS inhibitors and nonexposure to RAAS inhibitors was 1.161 (0.958-1.407) and 0.863 (0.533-1.397), respectively. When comparing exposure to RAAS inhibitors and nonexposure to RAAS inhibitors for intensive care unit admission, high-flow oxygen therapy, and death, the adjusted odds ratios (95% CIs) were 1.515 (0.402-5.701), 0.663 (0.272-1.619), and 1.363 (0.513-3.662), respectively. In all analyses, P values were not significant (P>0.05). The present study demonstrates the absence of an identifiable association between the exposure to RAAS inhibitors and risk and severity of COVID-19 infection, supporting the current medical guidelines and recommendations that patients should not discontinue RAAS inhibitors out of a concern that they are at increased risk for infection or severe illness of COVID-19. |*Angiotensin Receptor Antagonists/administration & dosage/adverse effects[MESH] |*Angiotensin-Converting Enzyme Inhibitors/administration & dosage/adverse effects[MESH] |*Coronavirus Infections/diagnosis/epidemiology/physiopathology[MESH] |*Hypertension/diagnosis/drug therapy/epidemiology[MESH] |*Pandemics[MESH] |*Pneumonia, Viral/diagnosis/epidemiology/physiopathology[MESH] |Betacoronavirus[MESH] |COVID-19[MESH] |Case-Control Studies[MESH] |Comorbidity[MESH] |Female[MESH] |Humans[MESH] |Male[MESH] |Middle Aged[MESH] |Prognosis[MESH] |Renin-Angiotensin System/drug effects[MESH] |Republic of Korea[MESH] |Retrospective Studies[MESH] |Risk Assessment[MESH] |SARS-CoV-2[MESH]Association Between Renin-Angiotensin-Aldosterone System Inhibitors an(epmc).pdf DeepDyve Pubget Overpricing