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10.1021/acschemneuro.0c00346

http://scihub22266oqcxt.onion/10.1021/acschemneuro.0c00346
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32605374!ä!32605374

suck abstract from ncbi

pmid32605374      ACS+Chem+Neurosci 2020 ; 11 (13): 1868-1870
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  • Cytokine Storm in COVID19: A Neural Hypothesis #MMPMID32605374
  • Ur A; Verma K
  • ACS Chem Neurosci 2020[Jul]; 11 (13): 1868-1870 PMID32605374show ga
  • Cytokine storm in COVID-19 is characterized by an excessive inflammatory response to SARS-CoV-2 that is caused by a dysregulated immune system of the host. We are proposing a new hypothesis that SARS-CoV-2 mediated inflammation of nucleus tractus solitarius (NTS) may be responsible for the cytokine storm in COVID 19. The inflamed NTS may result in a dysregulated cholinergic anti-inflammatory pathway and hypothalamic-pituitary-adrenal axis.
  • |Axons/immunology/metabolism/virology[MESH]
  • |Betacoronavirus/immunology/*metabolism[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/immunology/*metabolism[MESH]
  • |Cranial Nerves/immunology/metabolism/virology[MESH]
  • |Cytokines/immunology/*metabolism[MESH]
  • |Humans[MESH]
  • |Hypothalamo-Hypophyseal System/immunology/metabolism/virology[MESH]
  • |Inflammation Mediators/immunology/metabolism[MESH]
  • |Pandemics[MESH]
  • |Pituitary-Adrenal System/immunology/metabolism/virology[MESH]
  • |Pneumonia, Viral/immunology/*metabolism[MESH]
  • |SARS-CoV-2[MESH]


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