Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText.
Kick-your-searchterm to multiple Engines kick-your-query now !>
A dictionary by aggregated review articles of nephrology, medicine and the life sciences
Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

suck abstract from ncbi


10.1080/17843286.2020.1786324

http://scihub22266oqcxt.onion/10.1080/17843286.2020.1786324
suck pdf from google scholar
32597377!?!32597377

suck abstract from ncbi

pmid32597377      Acta+Clin+Belg 2022 ; 77 (1): 211-218
Nephropedia Template TP

gab.com Text

Twit Text FOAVip

Twit Text #

English Wikipedia


  • The role of angiotensin-converting enzyme 2 in the pathogenesis of COVID-19: the villain or the hero? #MMPMID32597377
  • Klhufek J
  • Acta Clin Belg 2022[Feb]; 77 (1): 211-218 PMID32597377show ga
  • Angiotensin-converting enzyme 2 (ACE 2) is the entry receptor for the novel coronavirus SARS-CoV-2, the aetiological agent of COVID-19. At the same time, ACE 2 expression decreases during COVID-19. Two seemingly contradictory relationships between the expression of ACE 2 and COVID-19 have been reported. Increased level of expression of ACE 2 may be a risk factor for the development of COVID-19 infection, while reduced ACE 2 expression during COVID-19 leads to acute respiratory distress syndrome. This article provides a comprehensive overview of available scientific knowledge about the role of ACE 2 in the pathogenesis of COVID-19, which is available up to current day. Also, it discusses unknown factors that we will have to reveal in order to understand the whole role of ACE 2 in the pathogenesis of COVID-19.
  • |*Angiotensin-Converting Enzyme 2[MESH]
  • |*COVID-19/pathology[MESH]
  • |Humans[MESH]
  • |Respiratory Distress Syndrome/virology[MESH]
  • |Risk Factors[MESH]


  • DeepDyve
  • Pubget Overpricing
  • suck abstract from ncbi

    Linkout box