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10.1155/2020/7413673 http://scihub22266oqcxt.onion/10.1155/2020/7413673 32596365!7254078!32596365     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32596365&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Biomed+Res+Int 2020 ; 2020 (?): 7413673 Nephropedia Template TP {{tp|p=32596365|t=2020. Changes of Laboratory Cardiac Markers and Mechanisms of Cardiac Injury in Coronavirus Disease 2019 |pdf=|usr=}}{{32596365}} gab.com Text Changes of Laboratory Cardiac Markers and Mechanisms of Cardiac Injury in Coronavirus Disease 2019 JO: Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=32596365 #FOAvip Some patients with coronavirus disease 2019 (COVID-19) show abnormal changes in laboratory myocardial injury markers, suggesting that patients with myocardial injury have a higher mortality rate than those without myocardial injury. This article reviews the possible mechanism of myocardial injury in patients with COVID-19. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) affects the patients with COVID-19 in aspects of direct infection of myocardial injury, specific binding to functional receptors on cardiomyocytes, and immune-mediated myocardial injury. During hospitalization, the monitoring of laboratory myocardial injury markers in patients of COVID-19 should be strengthened. Twit Text FOAVip Changes of Laboratory Cardiac Markers and Mechanisms of Cardiac Injury in Coronavirus Disease 2019 ????Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=32596365 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32596365 #FOAvip English Wikipedia <ref>{{Cite pmid|32596365}}</ref> Changes of Laboratory Cardiac Markers and Mechanisms of Cardiac Injury in Coronavirus Disease 2019 #MMPMID32596365 Li L; Zhou Q; Xu J Biomed Res Int 2020[]; 2020 (?): 7413673 PMID32596365show ga Some patients with coronavirus disease 2019 (COVID-19) show abnormal changes in laboratory myocardial injury markers, suggesting that patients with myocardial injury have a higher mortality rate than those without myocardial injury. This article reviews the possible mechanism of myocardial injury in patients with COVID-19. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) affects the patients with COVID-19 in aspects of direct infection of myocardial injury, specific binding to functional receptors on cardiomyocytes, and immune-mediated myocardial injury. During hospitalization, the monitoring of laboratory myocardial injury markers in patients of COVID-19 should be strengthened. |*Betacoronavirus[MESH] |Angiotensin-Converting Enzyme 2[MESH] |Biomarkers/blood/metabolism[MESH] |COVID-19[MESH] |Coronavirus Infections/*blood/*complications/metabolism[MESH] |Cytokines/blood/immunology[MESH] |Heart Injuries/*blood/*etiology/metabolism[MESH] |Humans[MESH] |Inflammation Mediators/blood/immunology[MESH] |Models, Cardiovascular[MESH] |Models, Immunological[MESH] |Myocytes, Cardiac/immunology/metabolism[MESH] |Pandemics[MESH] |Peptidyl-Dipeptidase A/metabolism[MESH] |Pneumonia, Viral/*blood/*complications/metabolism[MESH]Changes of Laboratory Cardiac Markers and Mechanisms of Cardiac Injury(epmc).pdf DeepDyve Pubget Overpricing