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10.1111/jth.14942 http://scihub22266oqcxt.onion/10.1111/jth.14942 32588535!7300603!32588535     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32588535&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Thromb+Haemost 2020 ; 18 (8): 1849-1852 Nephropedia Template TP {{tp|p=32588535|t=2020. The protective rather than prothrombotic fibrinogen in COVID-19 and other inflammatory states |pdf=|usr=}}{{32588535}} gab.com Text The protective rather than prothrombotic fibrinogen in COVID-19 and other inflammatory states JO: J Thromb Haemost http://www.kidney.de/pget.php?&tw=1&pmid=32588535 #FOAvip Hypercoagulability has been recognized as a common complication of COVID-19. Exact mechanisms for this extreme coagulation activation have not yet been elucidated. However, one of the consistent laboratory finding is the increase in fibrinogen, in some cases, marked elevation. High circulating levels of fibrinogen have been linked to thrombosis for years and for this reason, hyperfibrinogenemia is considered one of the mechanisms for COVID-19 coagulopathy. In this forum article, instead of the prothrombotic role, a protective function for fibrinogen is discussed. Fibrinogen, like the other well-known acute phase reactants, is increased in COVID-19 possibly to protect the host. Twit Text FOAVip The protective rather than prothrombotic fibrinogen in COVID-19 and other inflammatory states ????J Thromb Haemost http://www.kidney.de/pget.php?&tw=1&pmid=32588535 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32588535 #FOAvip English Wikipedia <ref>{{Cite pmid|32588535}}</ref> The protective rather than prothrombotic fibrinogen in COVID-19 and other inflammatory states #MMPMID32588535 Thachil J J Thromb Haemost 2020[Aug]; 18 (8): 1849-1852 PMID32588535show ga Hypercoagulability has been recognized as a common complication of COVID-19. Exact mechanisms for this extreme coagulation activation have not yet been elucidated. However, one of the consistent laboratory finding is the increase in fibrinogen, in some cases, marked elevation. High circulating levels of fibrinogen have been linked to thrombosis for years and for this reason, hyperfibrinogenemia is considered one of the mechanisms for COVID-19 coagulopathy. In this forum article, instead of the prothrombotic role, a protective function for fibrinogen is discussed. Fibrinogen, like the other well-known acute phase reactants, is increased in COVID-19 possibly to protect the host. |*Betacoronavirus[MESH] |Acute Disease[MESH] |Acute-Phase Proteins/physiology[MESH] |Biomarkers[MESH] |Blood Platelets/physiology[MESH] |COVID-19[MESH] |Coronavirus Infections/*blood/complications[MESH] |Fibrin Fibrinogen Degradation Products/analysis/physiology[MESH] |Fibrinogen/analysis/*physiology[MESH] |Humans[MESH] |Inflammation/*blood[MESH] |Models, Cardiovascular[MESH] |Pandemics[MESH] |Pneumonia, Viral/*blood/complications[MESH] |Risk[MESH] |SARS-CoV-2[MESH] |Thrombophilia/*blood/etiology[MESH] |Thrombosis/*prevention & control[MESH]The protective rather than prothrombotic fibrinogen in COVID-19 and ot(epmc).pdf DeepDyve Pubget Overpricing