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10.1111/dth.13871

http://scihub22266oqcxt.onion/10.1111/dth.13871
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32558055!7323108!32558055
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suck abstract from ncbi


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pmid32558055      Dermatol+Ther 2020 ; 33 (6): e13871
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  • SARS CoV-2 aggravates cellular metabolism mediated complications in COVID-19 infection #MMPMID32558055
  • Singh Y; Gupta G; Kazmi I; Al-Abbasi FA; Negi P; Chellappan DK; Dua K
  • Dermatol Ther 2020[Nov]; 33 (6): e13871 PMID32558055show ga
  • Severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2), the primary causative organism in corona virus disease-19 (COVID-19) infections, is a novel member of the human coronavirus family which was first identified in Wuhan, China, towards the end of 2019. This letter reveals new vital missing links in our current understanding of the mechanisms that lead to cell death triggered by ferroptotic stress in COVID-19 infection. It further reveal the importance of homocysteine mediated trans-sulfuration pathway in COVID-19 infection. Hence, Vitamin B6, folic acid, and Vitamin B12 should be incorporated in the treatment regimen for SARS CoV-2 infections to suppress complications, as the virus mediates altered host cell metabolism.
  • |COVID-19/*complications/prevention & control/virology[MESH]
  • |Cell Death/*physiology[MESH]
  • |Ferroptosis/*physiology[MESH]
  • |Folic Acid/administration & dosage[MESH]
  • |Humans[MESH]
  • |SARS-CoV-2/isolation & purification[MESH]
  • |Vitamin B 12/administration & dosage[MESH]


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