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10.1152/ajplung.00195.2020 http://scihub22266oqcxt.onion/10.1152/ajplung.00195.2020 32551862!7414237!32551862     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Physiol+Lung+Cell+Mol+Physiol 2020 ; 319 (2): L277-L288 Nephropedia Template TP {{tp|p=32551862|t=2020. Novel insights on the pulmonary vascular consequences of COVID-19 |pdf=|usr=}}{{32551862}} gab.com Text Novel insights on the pulmonary vascular consequences of COVID-19 JO: Am J Physiol Lung Cell Mol Physiol http://www.kidney.de/pget.php?&tw=1&pmid=32551862 #FOAvip In the last few months, the number of cases of a new coronavirus-related disease (COVID-19) rose exponentially, reaching the status of a pandemic. Interestingly, early imaging studies documented that pulmonary vascular thickening was specifically associated with COVID-19 pneumonia, implying a potential tropism of the virus for the pulmonary vasculature. Moreover, SARS-CoV-2 infection is associated with inflammation, hypoxia, oxidative stress, mitochondrial dysfunction, DNA damage, and lung coagulopathy promoting endothelial dysfunction and microthrombosis. These features are strikingly similar to what is seen in pulmonary vascular diseases. Although the consequences of COVID-19 on the pulmonary circulation remain to be explored, several viruses have been previously thought to be involved in the development of pulmonary vascular diseases. Patients with preexisting pulmonary vascular diseases also appear at increased risk of morbidity and mortality. The present article reviews the molecular factors shared by coronavirus infection and pulmonary vasculature defects, and the clinical relevance of pulmonary vascular alterations in the context of COVID-19. Twit Text FOAVip Novel insights on the pulmonary vascular consequences of COVID-19 ????Am J Physiol Lung Cell Mol Physiol http://www.kidney.de/pget.php?&tw=1&pmid=32551862 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32551862 #FOAvip English Wikipedia <ref>{{Cite pmid|32551862}}</ref> Novel insights on the pulmonary vascular consequences of COVID-19 #MMPMID32551862 Potus F; Mai V; Lebret M; Malenfant S; Breton-Gagnon E; Lajoie AC; Boucherat O; Bonnet S; Provencher S Am J Physiol Lung Cell Mol Physiol 2020[Aug]; 319 (2): L277-L288 PMID32551862show ga In the last few months, the number of cases of a new coronavirus-related disease (COVID-19) rose exponentially, reaching the status of a pandemic. Interestingly, early imaging studies documented that pulmonary vascular thickening was specifically associated with COVID-19 pneumonia, implying a potential tropism of the virus for the pulmonary vasculature. Moreover, SARS-CoV-2 infection is associated with inflammation, hypoxia, oxidative stress, mitochondrial dysfunction, DNA damage, and lung coagulopathy promoting endothelial dysfunction and microthrombosis. These features are strikingly similar to what is seen in pulmonary vascular diseases. Although the consequences of COVID-19 on the pulmonary circulation remain to be explored, several viruses have been previously thought to be involved in the development of pulmonary vascular diseases. Patients with preexisting pulmonary vascular diseases also appear at increased risk of morbidity and mortality. The present article reviews the molecular factors shared by coronavirus infection and pulmonary vasculature defects, and the clinical relevance of pulmonary vascular alterations in the context of COVID-19. |*Betacoronavirus/pathogenicity/physiology[MESH] |Angiotensin-Converting Enzyme 2[MESH] |COVID-19[MESH] |Coronavirus Infections/*complications/physiopathology/virology[MESH] |Cytokines/blood[MESH] |DNA Damage[MESH] |Heart Injuries/etiology[MESH] |Host Microbial Interactions/physiology[MESH] |Humans[MESH] |Hypoxia/etiology[MESH] |Inflammation Mediators/blood[MESH] |Lung Diseases/*etiology/physiopathology/virology[MESH] |Lung/*blood supply/*physiopathology/virology[MESH] |Mitochondria/physiology[MESH] |Myocardium[MESH] |Oxidative Stress[MESH] |Pandemics[MESH] |Peptidyl-Dipeptidase A/physiology[MESH] |Pneumonia, Viral/*complications/physiopathology/virology[MESH] |Pulmonary Circulation[MESH] |Pulmonary Embolism/etiology[MESH] |Receptors, Virus/physiology[MESH] |Risk Factors[MESH] |SARS-CoV-2[MESH]Novel insights on the pulmonary vascular consequences of COVID-19 (epmc).pdf DeepDyve Pubget Overpricing