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10.1016/j.psychres.2020.113198

http://scihub22266oqcxt.onion/10.1016/j.psychres.2020.113198
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32535509!ä!32535509

suck abstract from ncbi

pmid32535509      Psychiatry+Res 2020 ; 291 (ä): 113198
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  • COVID-19, interferons, and depression: A commentary #MMPMID32535509
  • Shader RI
  • Psychiatry Res 2020[Sep]; 291 (ä): 113198 PMID32535509show ga
  • An option currently being explored for the treatment of COVID-19 is the use of interferons (INFs), either alone or in combination with other antiviral agents. INFs are known to shift the metabolism of tryptophan (TRP) away from its role as a precursor of serotonin. For some patients, reduction in TRP levels may either expose an underlying vulnerability to depression or trigger a de novo episode of depression. This Commentary discusses the pathway involved and recommends in-hospital augmentation with foods or supplements that increase TRP levels for COVID-19 patients treated with INFs. Selective serotonin reuptake inhibitors may also be tried if the depressive symptomatology is not short-lived.
  • |*Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*metabolism/*psychology[MESH]
  • |Depression/*metabolism/psychology[MESH]
  • |Humans[MESH]
  • |Interferons/*metabolism[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*metabolism/*psychology[MESH]
  • |SARS-CoV-2[MESH]
  • |Serotonin/metabolism[MESH]


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