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10.1021/acschemneuro.0c00318

http://scihub22266oqcxt.onion/10.1021/acschemneuro.0c00318
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32530597!ä!32530597

suck abstract from ncbi

pmid32530597      ACS+Chem+Neurosci 2020 ; 11 (13): 1865-1867
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  • Happy Hypoxemia in COVID-19-A Neural Hypothesis #MMPMID32530597
  • U R A; Verma K
  • ACS Chem Neurosci 2020[Jul]; 11 (13): 1865-1867 PMID32530597show ga
  • Many COVID-19 patients are presenting with atypical clinical features. Happy hypoxemia with almost normal breathing, anosmia in the absence of rhinitis or nasal obstruction, and ageusia are some of the reported atypical clinical findings. Based on the clinical manifestations of the disease, we are proposing a new hypothesis that SARS-CoV-2 mediated inflammation of the nucleus tractus solitarius may be the reason for happy hypoxemia in COVID-19 patients.
  • |*Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/complications/*physiopathology[MESH]
  • |Cranial Nerves/physiopathology/virology[MESH]
  • |Humans[MESH]
  • |Hypoxia/etiology/*physiopathology[MESH]
  • |Inflammation/etiology/physiopathology/virology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/complications/*physiopathology[MESH]
  • |SARS-CoV-2[MESH]


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