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10.4149/gpb_2020019

http://scihub22266oqcxt.onion/10.4149/gpb_2020019
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32525813!ä!32525813

suck abstract from ncbi


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pmid32525813      Gen+Physiol+Biophys 2020 ; 39 (3): 203-204
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  • Renin-angiotensin system and SARS-CoV-2 interaction: underlying mechanisms and potential clinical implications #MMPMID32525813
  • Hrenak J; Zorad S; Simko F
  • Gen Physiol Biophys 2020[May]; 39 (3): 203-204 PMID32525813show ga
  • Renin-angiotensin system (RAS) inhibition supposedly increases the expression of angiotensin converting enzyme 2, serving as a binding site for SARS-CoV-2. Concerns arose regarding therapy with RAS inhibition during the COVID-19 pandemic. However, the pharmacological restraining the classical RAS axis might be beneficial due to the reduction of deleterious effects of angiotensin II and enhancement of the anti-inflammatory angiotensin 1-7 pathway. Unless large controlled studies are performed, RAS inhibition remains the cornerstone therapy in populations with cardiovascular disorders.
  • |*Renin-Angiotensin System[MESH]
  • |Angiotensin II Type 1 Receptor Blockers/therapeutic use[MESH]
  • |Angiotensin II/blood[MESH]
  • |Angiotensin-Converting Enzyme 2[MESH]
  • |Angiotensin-Converting Enzyme Inhibitors/therapeutic use[MESH]
  • |Betacoronavirus/pathogenicity/physiology[MESH]
  • |COVID-19[MESH]
  • |Cardiovascular Diseases/complications/*drug therapy[MESH]
  • |Coronavirus Infections/*complications/drug therapy[MESH]
  • |Humans[MESH]
  • |Pandemics[MESH]
  • |Peptidyl-Dipeptidase A[MESH]
  • |Pneumonia, Viral/*complications/drug therapy[MESH]
  • |SARS-CoV-2[MESH]


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