Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1021/acschemneuro.0c00249 http://scihub22266oqcxt.onion/10.1021/acschemneuro.0c00249 32525657!ä!32525657 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       ACS+Chem+Neurosci 2020 ; 11 (13): 1909-1913 Nephropedia Template TP {{tp|p=32525657|t=2020. Proinflammatory Cytokines in the Olfactory Mucosa Result in COVID-19 Induced Anosmia |pdf=|usr=}}{{32525657}} gab.com Text Proinflammatory Cytokines in the Olfactory Mucosa Result in COVID-19 Induced Anosmia JO: ACS Chem Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=32525657 #FOAvip Studies have found increased rates of dysosmia in patients with Novel Coronavirus disease 2019 (COVID-19). However, the mechanism that causes olfactory loss is unknown. The primary objective of this study was to explore local proinflammatory cytokine levels in the olfactory epithelium in patients with COVID-19. Biopsies of the olfactory epithelium were taken from patients with confirmed COVID-19 as well as uninfected controls. Levels of tumor necrosis factor alpha (TNF-alpha) and interleukin-1-beta (IL-1beta) were assessed using ELISA and compared between groups. Average TNF-alpha levels were significantly increased in the olfactory epithelium of the COVID-19 group compared to the control group (P < 0.05). However, no differences in IL-1beta were seen between groups. Elevated levels of the proinflammatory cytokine TNF-alpha were seen in the olfactory epithelium in patients with COVID-19. This suggests that direct inflammation of the olfactory epithelium could play a role in the acute olfactory loss described in many patients with COVID-19. Twit Text FOAVip Proinflammatory Cytokines in the Olfactory Mucosa Result in COVID-19 Induced Anosmia ????ACS Chem Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=32525657 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32525657 #FOAvip English Wikipedia <ref>{{Cite pmid|32525657}}</ref> Proinflammatory Cytokines in the Olfactory Mucosa Result in COVID-19 Induced Anosmia #MMPMID32525657 Torabi A; Mohammadbagheri E; Akbari Dilmaghani N; Bayat AH; Fathi M; Vakili K; Alizadeh R; Rezaeimirghaed O; Hajiesmaeili M; Ramezani M; Simani L; Aliaghaei A ACS Chem Neurosci 2020[Jul]; 11 (13): 1909-1913 PMID32525657show ga Studies have found increased rates of dysosmia in patients with Novel Coronavirus disease 2019 (COVID-19). However, the mechanism that causes olfactory loss is unknown. The primary objective of this study was to explore local proinflammatory cytokine levels in the olfactory epithelium in patients with COVID-19. Biopsies of the olfactory epithelium were taken from patients with confirmed COVID-19 as well as uninfected controls. Levels of tumor necrosis factor alpha (TNF-alpha) and interleukin-1-beta (IL-1beta) were assessed using ELISA and compared between groups. Average TNF-alpha levels were significantly increased in the olfactory epithelium of the COVID-19 group compared to the control group (P < 0.05). However, no differences in IL-1beta were seen between groups. Elevated levels of the proinflammatory cytokine TNF-alpha were seen in the olfactory epithelium in patients with COVID-19. This suggests that direct inflammation of the olfactory epithelium could play a role in the acute olfactory loss described in many patients with COVID-19. |*Betacoronavirus[MESH] |Adult[MESH] |COVID-19[MESH] |Coronavirus Infections/complications/diagnosis/*metabolism[MESH] |Cytokines/analysis/*metabolism[MESH] |Female[MESH] |Humans[MESH] |Male[MESH] |Middle Aged[MESH] |Olfaction Disorders/diagnosis/etiology/*metabolism[MESH] |Olfactory Mucosa/*metabolism[MESH] |Pandemics[MESH] |Pneumonia, Viral/complications/diagnosis/*metabolism[MESH] |SARS-CoV-2[MESH]Proinflammatory Cytokines in the Olfactory Mucosa Result in COVID-19 I(epmc).pdf DeepDyve Pubget Overpricing