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10.1016/j.hjc.2020.05.007 http://scihub22266oqcxt.onion/10.1016/j.hjc.2020.05.007 32522617!7275139!32522617     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Hellenic+J+Cardiol 2021 ; 62 (1): 13-23 Nephropedia Template TP {{tp|p=32522617|t=2021. Outbreak of SARS-CoV2: Pathogenesis of infection and cardiovascular involvement |pdf=|usr=}}{{32522617}} gab.com Text Outbreak of SARS-CoV2: Pathogenesis of infection and cardiovascular involvement JO: Hellenic J Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=32522617 #FOAvip Since the new severe acute respiratory syndrome coronavirus 2 (SARS-CoV2) has emerged from China, the infection (novel corona virus disease-2019, COVID-19) has affected many countries and led to many deaths worldwide. Like SARS-CoV, angiotencin converting enzyme (ACE)2 as a functional receptor for SARS-CoV2 is essential for the virus to make an entry into the cell. ACE2 is a part of Renin-Angiotensin-Aldosterone System, which is expressed in several organs that opposes the angiotensin (Ang) II functions by converting Ang II to Ang (1-7), the one with vasodilation effects. The death rate of COVID-19 is estimated to be approximately 3.4%; however, some comorbid conditions like underlying cardiovascular disease, hypertension, and diabetes increase the risk of mortality. In addition, cardiovascular involvement as a complication of SARS-CoV2 could be direct through either ACE2 receptors that are expressed tremendously in the heart, or by the surge of different cytokines or by acute respiratory distress syndrome-induced hypoxia. Traditional risk factors could aggravate the process of COVID-19 infection that urges the triage of these high-risk patients for SARS-CoV2. Currently, there is no effective, proven treatment or vaccination for COVID-19, but many investigators are struggling to find a treatment strategy as soon as possible. Some potential medications like chloroquine by itself or in combination with azithromycin and some protease inhibitors used for the treatment of COVID-19 have cardiovascular adverse effects, which should be kept in mind while the patients taking these medications are being closely monitored. Twit Text FOAVip Outbreak of SARS-CoV2: Pathogenesis of infection and cardiovascular involvement ????Hellenic J Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=32522617 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32522617 #FOAvip English Wikipedia <ref>{{Cite pmid|32522617}}</ref> Outbreak of SARS-CoV2: Pathogenesis of infection and cardiovascular involvement #MMPMID32522617 Amirfakhryan H; Safari F Hellenic J Cardiol 2021[Jan]; 62 (1): 13-23 PMID32522617show ga Since the new severe acute respiratory syndrome coronavirus 2 (SARS-CoV2) has emerged from China, the infection (novel corona virus disease-2019, COVID-19) has affected many countries and led to many deaths worldwide. Like SARS-CoV, angiotencin converting enzyme (ACE)2 as a functional receptor for SARS-CoV2 is essential for the virus to make an entry into the cell. ACE2 is a part of Renin-Angiotensin-Aldosterone System, which is expressed in several organs that opposes the angiotensin (Ang) II functions by converting Ang II to Ang (1-7), the one with vasodilation effects. The death rate of COVID-19 is estimated to be approximately 3.4%; however, some comorbid conditions like underlying cardiovascular disease, hypertension, and diabetes increase the risk of mortality. In addition, cardiovascular involvement as a complication of SARS-CoV2 could be direct through either ACE2 receptors that are expressed tremendously in the heart, or by the surge of different cytokines or by acute respiratory distress syndrome-induced hypoxia. Traditional risk factors could aggravate the process of COVID-19 infection that urges the triage of these high-risk patients for SARS-CoV2. Currently, there is no effective, proven treatment or vaccination for COVID-19, but many investigators are struggling to find a treatment strategy as soon as possible. Some potential medications like chloroquine by itself or in combination with azithromycin and some protease inhibitors used for the treatment of COVID-19 have cardiovascular adverse effects, which should be kept in mind while the patients taking these medications are being closely monitored. |*COVID-19 Drug Treatment[MESH] |*COVID-19/epidemiology/therapy/virology[MESH] |*Cardiovascular Diseases/epidemiology/metabolism/physiopathology[MESH] |*SARS-CoV-2/pathogenicity/physiology[MESH] |Angiotensin-Converting Enzyme 2/*metabolism[MESH] |Humans[MESH] |Peptidyl-Dipeptidase A/*metabolism[MESH] |Risk Factors[MESH]Outbreak of SARS-CoV2: Pathogenesis of infection and cardiovascular in(epmc).pdf DeepDyve Pubget Overpricing