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10.1016/j.cytogfr.2020.06.001 http://scihub22266oqcxt.onion/10.1016/j.cytogfr.2020.06.001 32513566!7265853!32513566     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cytokine+Growth+Factor+Rev 2020 ; 54 (ä): 62-75 Nephropedia Template TP {{tp|p=32513566|t=2020. SARS-CoV-2 infection: The role of cytokines in COVID-19 disease |pdf=|usr=}}{{32513566}} gab.com Text SARS-CoV-2 infection: The role of cytokines in COVID-19 disease JO: Cytokine Growth Factor Rev http://www.kidney.de/pget.php?&tw=1&pmid=32513566 #FOAvip COVID-19 disease, caused by infection with SARS-CoV-2, is related to a series of physiopathological mechanisms that mobilize a wide variety of biomolecules, mainly immunological in nature. In the most severe cases, the prognosis can be markedly worsened by the hyperproduction of mainly proinflammatory cytokines, such as IL-1, IL-6, IL-12, IFN-gamma, and TNF-alpha, preferentially targeting lung tissue. This study reviews published data on alterations in the expression of different cytokines in patients with COVID-19 who require admission to an intensive care unit. Data on the implication of cytokines in this disease and their effect on outcomes will support the design of more effective approaches to the management of COVID-19. Twit Text FOAVip SARS-CoV-2 infection: The role of cytokines in COVID-19 disease ????Cytokine Growth Factor Rev http://www.kidney.de/pget.php?&tw=1&pmid=32513566 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32513566 #FOAvip English Wikipedia <ref>{{Cite pmid|32513566}}</ref> SARS-CoV-2 infection: The role of cytokines in COVID-19 disease #MMPMID32513566 Costela-Ruiz VJ; Illescas-Montes R; Puerta-Puerta JM; Ruiz C; Melguizo-Rodriguez L Cytokine Growth Factor Rev 2020[Aug]; 54 (ä): 62-75 PMID32513566show ga COVID-19 disease, caused by infection with SARS-CoV-2, is related to a series of physiopathological mechanisms that mobilize a wide variety of biomolecules, mainly immunological in nature. In the most severe cases, the prognosis can be markedly worsened by the hyperproduction of mainly proinflammatory cytokines, such as IL-1, IL-6, IL-12, IFN-gamma, and TNF-alpha, preferentially targeting lung tissue. This study reviews published data on alterations in the expression of different cytokines in patients with COVID-19 who require admission to an intensive care unit. Data on the implication of cytokines in this disease and their effect on outcomes will support the design of more effective approaches to the management of COVID-19. |Betacoronavirus/*immunology[MESH] |COVID-19[MESH] |Coronavirus Infections/immunology/mortality/*pathology[MESH] |Cytokine Release Syndrome/immunology/*pathology[MESH] |Cytokines/*metabolism[MESH] |Humans[MESH] |Pandemics[MESH] |Pneumonia, Viral/immunology/mortality/*pathology[MESH] |SARS-CoV-2[MESH]SARS-CoV-2 infection: The role of cytokines in COVID-19 disease (epmc).pdf DeepDyve Pubget Overpricing