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10.1111/tid.13355 http://scihub22266oqcxt.onion/10.1111/tid.13355 32510756!ä!32510756 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 267.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Transpl+Infect+Dis 2020 ; 22 (6): e13355 Nephropedia Template TP {{tp|p=32510756|t=2020. Hyponatremia: A possible immuno-neuroendocrine interface with COVID-19 in a kidney transplant recipient |pdf=|usr=}}{{32510756}} gab.com Text Hyponatremia: A possible immuno-neuroendocrine interface with COVID-19 in a kidney transplant recipient JO: Transpl Infect Dis http://www.kidney.de/pget.php?&tw=1&pmid=32510756 #FOAvip There is fast-emerging, cumulative clinical data on coronavirus disease 2019 (COVID-19) in kidney transplant recipients. Although respiratory tract symptoms are often the initial presentation among kidney transplant recipients who contract COVID-19, other clinical features which may indicate underlying SARS-CoV-2-related inflammation, such as gastrointestinal symptoms, are not uncommon. Hyponatremia can develop and may reflect underlying inflammation. Interferon-6 is an important pro-inflammatory cytokine involved in the pathogenesis of severe COVID-19 complications and may play a role in the inappropriately higher secretion of antidiuretic hormone leading to hyponatremia. This pathway is the so-called immuno-neuroendocrine interface. Hyponatremia in COVID-19 has been reported in a few case series of non-kidney transplant patients and only one reported kidney transplant recipient. However, the clinical course and prognostic value of hyponatremia in this population are not described in detail. We report a kidney transplant recipient who was infected with COVID-19 and exhibited severe hyponatremia secondary to the syndrome of inappropriate antidiuretic hormone secretion. Hyponatremia is one of the clinical presentations of COVID-19, although less common, and may occur more frequently in kidney transplant recipients. Thus, the possible underlying immuno-neuroendocrine relationship related to the inflammatory process of COVID-19 leading to hyponatremia and its prognostic value are reviewed. Twit Text FOAVip Hyponatremia: A possible immuno-neuroendocrine interface with COVID-19 in a kidney transplant recipient ????Transpl Infect Dis http://www.kidney.de/pget.php?&tw=1&pmid=32510756 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32510756 #FOAvip English Wikipedia <ref>{{Cite pmid|32510756}}</ref> Hyponatremia: A possible immuno-neuroendocrine interface with COVID-19 in a kidney transplant recipient #MMPMID32510756 Tantisattamo E; Reddy UG; Duong DK; Ferrey AJ; Ichii H; Dafoe DC; Kalantar-Zadeh K Transpl Infect Dis 2020[Dec]; 22 (6): e13355 PMID32510756show ga There is fast-emerging, cumulative clinical data on coronavirus disease 2019 (COVID-19) in kidney transplant recipients. Although respiratory tract symptoms are often the initial presentation among kidney transplant recipients who contract COVID-19, other clinical features which may indicate underlying SARS-CoV-2-related inflammation, such as gastrointestinal symptoms, are not uncommon. Hyponatremia can develop and may reflect underlying inflammation. Interferon-6 is an important pro-inflammatory cytokine involved in the pathogenesis of severe COVID-19 complications and may play a role in the inappropriately higher secretion of antidiuretic hormone leading to hyponatremia. This pathway is the so-called immuno-neuroendocrine interface. Hyponatremia in COVID-19 has been reported in a few case series of non-kidney transplant patients and only one reported kidney transplant recipient. However, the clinical course and prognostic value of hyponatremia in this population are not described in detail. We report a kidney transplant recipient who was infected with COVID-19 and exhibited severe hyponatremia secondary to the syndrome of inappropriate antidiuretic hormone secretion. Hyponatremia is one of the clinical presentations of COVID-19, although less common, and may occur more frequently in kidney transplant recipients. Thus, the possible underlying immuno-neuroendocrine relationship related to the inflammatory process of COVID-19 leading to hyponatremia and its prognostic value are reviewed. |*Kidney Transplantation[MESH] |COVID-19/*immunology/metabolism[MESH] |Female[MESH] |Graft Rejection/prevention & control[MESH] |Humans[MESH] |Hyponatremia/*immunology/metabolism[MESH] |Immunosuppressive Agents/*therapeutic use[MESH] |Inappropriate ADH Syndrome/*immunology/metabolism[MESH] |Middle Aged[MESH] |Neuroimmunomodulation/immunology[MESH] |Neurosecretory Systems/immunology[MESH]Hyponatremia: A possible immuno-neuroendocrine interface with COVID-19(epmc).pdf DeepDyve Pubget Overpricing