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10.1136/heartjnl-2020-317186 http://scihub22266oqcxt.onion/10.1136/heartjnl-2020-317186 32499236!ä!32499236 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Heart 2020 ; 106 (15): 1127-1131 Nephropedia Template TP {{tp|p=32499236|t=2020. COVID-19 pandemic and troponin: indirect myocardial injury, myocardial inflammation or myocarditis?|pdf=|usr=}}{{32499236}} gab.com Text COVID-19 pandemic and troponin: indirect myocardial injury, myocardial inflammation or myocarditis JO: Heart http://www.kidney.de/pget.php?&tw=1&pmid=32499236 #FOAvip The initial mechanism for severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infection is the binding of the virus to the membrane-bound form of ACE2, which is mainly expressed in the lung. Since the heart and the vessels also express ACE2, they both could become targets of the virus. However, at present the extent and importance of this potential involvement are unknown. Cardiac troponin levels are significantly higher in patients with more severe infections, patients admitted to intensive care units or in those who have died. In the setting of COVID-19, myocardial injury, defined by an increased troponin level, occurs especially due to non-ischaemic myocardial processes, including severe respiratory infection with hypoxia, sepsis, systemic inflammation, pulmonary thrombosis and embolism, cardiac adrenergic hyperstimulation during cytokine storm syndrome, and myocarditis. At present, there are limited reports on definite diagnosis of myocarditis caused by SARS-CoV-2 in humans and limited demonstration of the virus in the myocardium. In conclusion, although the heart and the vessels are potential targets in COVID-19, there is currently limited evidence on the direct infection of the myocardium by SARS-CoV-2. Additional pathological studies and autopsy series will be very helpful to clarify the potentiality of COVID-19 to directly infect the myocardium and cause myocarditis. Twit Text FOAVip COVID-19 pandemic and troponin: indirect myocardial injury, myocardial inflammation or myocarditis ????Heart http://www.kidney.de/pget.php?&tw=1&pmid=32499236 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32499236 #FOAvip English Wikipedia <ref>{{Cite pmid|32499236}}</ref> COVID-19 pandemic and troponin: indirect myocardial injury, myocardial inflammation or myocarditis? #MMPMID32499236 Imazio M; Klingel K; Kindermann I; Brucato A; De Rosa FG; Adler Y; De Ferrari GM Heart 2020[Aug]; 106 (15): 1127-1131 PMID32499236show ga The initial mechanism for severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infection is the binding of the virus to the membrane-bound form of ACE2, which is mainly expressed in the lung. Since the heart and the vessels also express ACE2, they both could become targets of the virus. However, at present the extent and importance of this potential involvement are unknown. Cardiac troponin levels are significantly higher in patients with more severe infections, patients admitted to intensive care units or in those who have died. In the setting of COVID-19, myocardial injury, defined by an increased troponin level, occurs especially due to non-ischaemic myocardial processes, including severe respiratory infection with hypoxia, sepsis, systemic inflammation, pulmonary thrombosis and embolism, cardiac adrenergic hyperstimulation during cytokine storm syndrome, and myocarditis. At present, there are limited reports on definite diagnosis of myocarditis caused by SARS-CoV-2 in humans and limited demonstration of the virus in the myocardium. In conclusion, although the heart and the vessels are potential targets in COVID-19, there is currently limited evidence on the direct infection of the myocardium by SARS-CoV-2. Additional pathological studies and autopsy series will be very helpful to clarify the potentiality of COVID-19 to directly infect the myocardium and cause myocarditis. |Angiotensin-Converting Enzyme 2[MESH] |Betacoronavirus[MESH] |Biomarkers/blood[MESH] |COVID-19[MESH] |Coronavirus Infections/epidemiology/*virology[MESH] |Humans[MESH] |Inflammation/*virology[MESH] |Myocarditis/*virology[MESH] |Pandemics[MESH] |Peptidyl-Dipeptidase A/metabolism[MESH] |Pneumonia, Viral/epidemiology/*virology[MESH] |Renin-Angiotensin System/physiology[MESH] |SARS-CoV-2[MESH]COVID-19 pandemic and troponin: indirect myocardial injury, myocardial(epmc).pdf DeepDyve Pubget Overpricing