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10.2147/VHRM.S242685 http://scihub22266oqcxt.onion/10.2147/VHRM.S242685 32494148!7229867!32494148     free     free     free Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Vasc+Health+Risk+Manag 2020 ; 16 (ä): 167-180 Nephropedia Template TP {{tp|p=32494148|t=2020. Vascular Calcification: An Important Understanding in Nephrology |pdf=|usr=}}{{32494148}} gab.com Text Vascular Calcification: An Important Understanding in Nephrology JO: Vasc Health Risk Manag http://www.kidney.de/pget.php?&tw=1&pmid=32494148 #FOAvip Vascular calcification (VC) is a life-threatening state in chronic kidney disease (CKD). ?High cardiovascular mortality and morbidity of CKD cases may root from medial VC promoted by hyperphosphatemia. Vascular calcification is an active, highly regulated, and complex biological process that is mediated by genetics, epigenetics, dysregulated form of matrix mineral metabolism, hormones, and the activation of cellular signaling pathways. Moreover, gut microbiome as a source of uremic toxins (eg, phosphate, advanced glycation end products and indoxyl-sulfate) can be regarded as a potential contributor to VC in CKD. Here, an update on different cellular and molecular processes involved in VC in CKD is discussed to elucidate the probable therapeutic pathways in the future. Twit Text FOAVip Vascular Calcification: An Important Understanding in Nephrology ????Vasc Health Risk Manag http://www.kidney.de/pget.php?&tw=1&pmid=32494148 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32494148 #FOAvip English Wikipedia <ref>{{Cite pmid|32494148}}</ref> Vascular Calcification: An Important Understanding in Nephrology #MMPMID32494148 Zununi Vahed S; Mostafavi S; Hosseiniyan Khatibi SM; Shoja MM; Ardalan M Vasc Health Risk Manag 2020[]; 16 (ä): 167-180 PMID32494148show ga Vascular calcification (VC) is a life-threatening state in chronic kidney disease (CKD). ?High cardiovascular mortality and morbidity of CKD cases may root from medial VC promoted by hyperphosphatemia. Vascular calcification is an active, highly regulated, and complex biological process that is mediated by genetics, epigenetics, dysregulated form of matrix mineral metabolism, hormones, and the activation of cellular signaling pathways. Moreover, gut microbiome as a source of uremic toxins (eg, phosphate, advanced glycation end products and indoxyl-sulfate) can be regarded as a potential contributor to VC in CKD. Here, an update on different cellular and molecular processes involved in VC in CKD is discussed to elucidate the probable therapeutic pathways in the future. |*Renal Elimination[MESH] |*Vascular Remodeling[MESH] |Animals[MESH] |Blood Vessels/*metabolism/pathology/physiopathology[MESH] |Humans[MESH] |Kidney/*metabolism/physiopathology[MESH] |Prognosis[MESH] |Renal Insufficiency, Chronic/*complications/metabolism/mortality/physiopathology[MESH] |Risk Factors[MESH]Vascular Calcification: An Important Understanding in Nephrology (epmc).pdf DeepDyve Pubget Overpricing