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10.4049/jimmunol.2000513

http://scihub22266oqcxt.onion/10.4049/jimmunol.2000513
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32493814!7343621!32493814
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suck abstract from ncbi


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pmid32493814      J+Immunol 2020 ; 205 (2): 307-312
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  • Inflammasomes and Pyroptosis as Therapeutic Targets for COVID-19 #MMPMID32493814
  • Yap JKY; Moriyama M; Iwasaki A
  • J Immunol 2020[Jul]; 205 (2): 307-312 PMID32493814show ga
  • The inflammatory response to severe acute respiratory syndrome-related coronavirus 2 infection has a direct impact on the clinical outcomes of coronavirus disease 2019 patients. Of the many innate immune pathways that are engaged by severe acute respiratory syndrome-related coronavirus 2, we highlight the importance of the inflammasome pathway. We discuss available pharmaceutical agents that target a critical component of inflammasome activation, signaling leading to cellular pyroptosis, and the downstream cytokines as a promising target for the treatment of severe coronavirus disease 2019-associated diseases.
  • |Animals[MESH]
  • |Antiviral Agents/immunology/*pharmacology[MESH]
  • |Betacoronavirus/physiology[MESH]
  • |COVID-19[MESH]
  • |COVID-19 Drug Treatment[MESH]
  • |Coronavirus Infections/drug therapy/immunology/pathology[MESH]
  • |Humans[MESH]
  • |Immunity, Innate[MESH]
  • |Inflammasomes/*drug effects[MESH]
  • |Intercellular Signaling Peptides and Proteins/metabolism[MESH]
  • |Macrophages, Alveolar/pathology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/immunology/pathology[MESH]
  • |Pyroptosis/*drug effects[MESH]
  • |SARS-CoV-2[MESH]
  • |Severe acute respiratory syndrome-related coronavirus/physiology[MESH]


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