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suck abstract from ncbi

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  • Thromboinflammatory response in SARS-CoV-2 sepsis #MMPMID32490726
  • Maiese A; Passaro G; Matteis A; Fazio V; Raffaele R; Paolo MD
  • Med Leg J 2020[Jul]; 88 (2): 78-80 PMID32490726show ga
  • Viral sepsis is rare, and its real incidence is not known. SARS-CoV-2 infection causes the release of a significant amount of pro-inflammatory cytokines that aggravates interstitial pneumonia and evolves in viral sepsis with prominent hypercoagulability. We believe it is useful and advisable to establish early immunomodulator therapy and the prophylaxis anticoagulant therapy should be rethought.
  • |*Betacoronavirus[MESH]
  • |Age Factors[MESH]
  • |Anticoagulants/therapeutic use[MESH]
  • |Biomarkers/blood[MESH]
  • |Comorbidity[MESH]
  • |Coronavirus Infections/*complications/drug therapy[MESH]
  • |Cytokines/*blood[MESH]
  • |Fibrin Fibrinogen Degradation Products/analysis[MESH]
  • |Humans[MESH]
  • |Immunosuppressive Agents/therapeutic use[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*complications/drug therapy[MESH]
  • |Sepsis/blood/*virology[MESH]
  • |Thrombosis/prevention & control/*virology[MESH]

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  • suck abstract from ncbi

    78 2.88 2020