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10.1038/s41577-020-0354-x

http://scihub22266oqcxt.onion/10.1038/s41577-020-0354-x
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suck abstract from ncbi


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pmid32488201      Nat+Rev+Immunol 2020 ; 20 (7): 395-396
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  • Blood clots and TAM receptor signalling in COVID-19 pathogenesis #MMPMID32488201
  • Lemke G; Silverman GJ
  • Nat Rev Immunol 2020[Jul]; 20 (7): 395-396 PMID32488201show ga
  • We suggest that the exuberant blood clotting and immune hyper-reaction seen in patients with COVID-19 may be exacerbated by depletion of the same regulator. This agent is protein S, which is both an anticoagulant in the blood coagulation cascade and an activating ligand for the immunosuppressive TAM family of receptor tyrosine kinases.
  • |Animals[MESH]
  • |Betacoronavirus[MESH]
  • |COVID-19[MESH]
  • |Calcium-Binding Proteins/metabolism[MESH]
  • |Coronavirus Infections/immunology/metabolism/*pathology[MESH]
  • |Cytokine Release Syndrome/pathology[MESH]
  • |Humans[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/immunology/metabolism/*pathology[MESH]
  • |Protein S[MESH]
  • |Receptor Protein-Tyrosine Kinases/metabolism[MESH]
  • |SARS-CoV-2[MESH]
  • |Signal Transduction[MESH]


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