Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1002/cbin.11403 http://scihub22266oqcxt.onion/10.1002/cbin.11403 32458561!7283672!32458561     free     free     free       Cell+Biol+Int 2020 ; 44 (9): 1792-1797 Nephropedia Template TP {{tp|p=32458561|t=2020. Lymphopenia in COVID-19: Therapeutic opportunities |pdf=|usr=}}{{32458561}} gab.com Text Lymphopenia in COVID-19: Therapeutic opportunities JO: Cell Biol Int http://www.kidney.de/pget.php?&tw=1&pmid=32458561 #FOAvip Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is uncontrollably spread all over the world. The host immune responses strongly try to confront it with all the potential cells and cytokines. With chronically condition of SARS-CoV-2, natural killer cells and T cells become exhausted and decreasing their count leads to lymphopenia. Inability to eradicate the infected organ makes hyperinitiation of the immune system, which releases the excessive inflammatory cytokines to compensate the exhausted one as well as the low lymphocytes counts; it consequently leads to the cytokine storm syndrome. These mechanisms and the potential therapeutic targeting are discussed in this paper. Twit Text FOAVip Lymphopenia in COVID-19: Therapeutic opportunities ????Cell Biol Int http://www.kidney.de/pget.php?&tw=1&pmid=32458561 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32458561 #FOAvip English Wikipedia <ref>{{Cite pmid|32458561}}</ref> Lymphopenia in COVID-19: Therapeutic opportunities #MMPMID32458561 Fathi N; Rezaei N Cell Biol Int 2020[Sep]; 44 (9): 1792-1797 PMID32458561show ga Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is uncontrollably spread all over the world. The host immune responses strongly try to confront it with all the potential cells and cytokines. With chronically condition of SARS-CoV-2, natural killer cells and T cells become exhausted and decreasing their count leads to lymphopenia. Inability to eradicate the infected organ makes hyperinitiation of the immune system, which releases the excessive inflammatory cytokines to compensate the exhausted one as well as the low lymphocytes counts; it consequently leads to the cytokine storm syndrome. These mechanisms and the potential therapeutic targeting are discussed in this paper. |Betacoronavirus/*immunology[MESH] |COVID-19[MESH] |Coronavirus Infections/epidemiology/*immunology/*therapy[MESH] |Cytokines/antagonists & inhibitors/immunology[MESH] |Humans[MESH] |Immunosuppressive Agents/administration & dosage[MESH] |Immunotherapy/methods[MESH] |Killer Cells, Natural/drug effects/immunology[MESH] |Lymphopenia/epidemiology/*immunology/*therapy[MESH] |Pandemics[MESH] |Pneumonia, Viral/epidemiology/*immunology/*therapy[MESH] |SARS-CoV-2[MESH]Lymphopenia in COVID-19: Therapeutic opportunities (epmc).pdf DeepDyve Pubget Overpricing