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10.1016/j.diabres.2020.108217 http://scihub22266oqcxt.onion/10.1016/j.diabres.2020.108217 32451317!7217793!32451317     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Diabetes+Res+Clin+Pract 2020 ; 164 (ä): 108217 Nephropedia Template TP {{tp|p=32451317|t=2020. The contribution of diabetic micro-angiopathy to adverse outcomes in COVID-19 |pdf=|usr=}}{{32451317}} gab.com Text The contribution of diabetic micro-angiopathy to adverse outcomes in COVID-19 JO: Diabetes Res Clin Pract http://www.kidney.de/pget.php?&tw=1&pmid=32451317 #FOAvip Increasing evidence points to endothelial cell dysfunction as a key pathophysiological factor in severe coronavirus disease-19 (COVID-19), manifested by platelet aggregation, microthrombi and altered vasomotor tone. This may be driven by direct endothelial cell entry by the virus, or indirectly by activated inflammatory cascade. Major risk groups identified for adverse outcomes in COVID-19 are diabetes, and those from the Black, Asian and ethnic minority (BAME) populations. Hyperglycaemia (expressed as glycated haemoglobin or mean hospital glucose) correlates with worse outcomes in COVID-19. It is not known whether hyperglycaemia is causative or is a surrogate marker - persistent hyperglycaemia is well known as an aetiological agent in microangiopathy. In this article, we propose that pre-existing endothelial dysfunction of microangiopathy, more commonly evident in diabetes and BAME groups, makes an individual vulnerable to the subsequent 'endothelitis' of COVID-19 infection. Twit Text FOAVip The contribution of diabetic micro-angiopathy to adverse outcomes in COVID-19 ????Diabetes Res Clin Pract http://www.kidney.de/pget.php?&tw=1&pmid=32451317 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32451317 #FOAvip English Wikipedia <ref>{{Cite pmid|32451317}}</ref> The contribution of diabetic micro-angiopathy to adverse outcomes in COVID-19 #MMPMID32451317 Whyte MB; Vas P; Heiss C; Feher MD Diabetes Res Clin Pract 2020[Jun]; 164 (ä): 108217 PMID32451317show ga Increasing evidence points to endothelial cell dysfunction as a key pathophysiological factor in severe coronavirus disease-19 (COVID-19), manifested by platelet aggregation, microthrombi and altered vasomotor tone. This may be driven by direct endothelial cell entry by the virus, or indirectly by activated inflammatory cascade. Major risk groups identified for adverse outcomes in COVID-19 are diabetes, and those from the Black, Asian and ethnic minority (BAME) populations. Hyperglycaemia (expressed as glycated haemoglobin or mean hospital glucose) correlates with worse outcomes in COVID-19. It is not known whether hyperglycaemia is causative or is a surrogate marker - persistent hyperglycaemia is well known as an aetiological agent in microangiopathy. In this article, we propose that pre-existing endothelial dysfunction of microangiopathy, more commonly evident in diabetes and BAME groups, makes an individual vulnerable to the subsequent 'endothelitis' of COVID-19 infection. |Betacoronavirus/isolation & purification[MESH] |COVID-19[MESH] |Coronavirus Infections/blood/*pathology/therapy[MESH] |Diabetic Angiopathies/pathology/*virology[MESH] |Ethnicity[MESH] |Humans[MESH] |Hyperglycemia/pathology/virology[MESH] |Pandemics[MESH] |Pneumonia, Viral/blood/*pathology/therapy[MESH]The contribution of diabetic micro-angiopathy to adverse outcomes in C(epmc).pdf DeepDyve Pubget Overpricing