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10.1016/j.dsx.2020.05.022

http://scihub22266oqcxt.onion/10.1016/j.dsx.2020.05.022
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32428864!7214324!32428864
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suck abstract from ncbi


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pmid32428864      Diabetes+Metab+Syndr 2020 ; 14 (4): 637-639
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  • Angiotensin converting enzyme-2 as therapeutic target in COVID-19 #MMPMID32428864
  • Roshanravan N; Ghaffari S; Hedayati M
  • Diabetes Metab Syndr 2020[Jul]; 14 (4): 637-639 PMID32428864show ga
  • The pandemic of coronavirus disease 2019 (COVID-19) is a global health emergency that poses a significant threat to world people's health. This outbreak causes major challenges to healthcare systems. Given the lack of effective treatments or vaccine for it, the identification of novel and safe drugs against COVID-19 infection is an urgent need. Angiotensin-converting enzyme 2 (ACE2) is not only an entry receptor of the SARS-CoV-2 virus, the virus that causes COVID-19, but also can protect from lung injury. In this view, we highlighted potential approaches to address ACE2-mediated SARS-CoV-2 virus, including 1) delivering an excessive soluble form of ACE2 (recombinant human ACE2: rhACE2) and 2) inhibition of the interaction between SARS-CoV-2 virus and ACE2 by some compounds with competitive effects (morphine and codeine). Further clinical trials in this regard can reveal a more definite conclusion against the COVID-19 disaster.
  • |*Molecular Targeted Therapy[MESH]
  • |Angiotensin-Converting Enzyme 2[MESH]
  • |Angiotensin-Converting Enzyme Inhibitors/*therapeutic use[MESH]
  • |Betacoronavirus/*drug effects/genetics[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*drug therapy/*enzymology/genetics[MESH]
  • |Gene Expression Regulation[MESH]
  • |Host-Pathogen Interactions/*drug effects/genetics[MESH]
  • |Humans[MESH]
  • |Pandemics[MESH]
  • |Peptidyl-Dipeptidase A/*metabolism/therapeutic use[MESH]
  • |Pneumonia, Viral/*drug therapy/*enzymology/genetics[MESH]
  • |Receptors, Virus/antagonists & inhibitors/metabolism[MESH]
  • |SARS-CoV-2[MESH]


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