| Exercise as medicine for COVID-19: An ACE in the hole? #MMPMID32428811Heffernan KS; Jae SYMed Hypotheses 2020[Sep]; 142 (ä): 109835 PMID32428811show ga
ä|*Betacoronavirus/physiology[MESH]|Angiotensin I/physiology[MESH]|Angiotensin Receptor Antagonists/adverse effects[MESH]|Angiotensin-Converting Enzyme 2[MESH]|Angiotensin-Converting Enzyme Inhibitors/adverse effects[MESH]|COVID-19[MESH]|Coronavirus Infections/etiology/physiopathology/*prevention & control[MESH]|Disease Susceptibility[MESH]|Exercise/*physiology[MESH]|Host Microbial Interactions/physiology[MESH]|Humans[MESH]|Models, Biological[MESH]|Pandemics/*prevention & control[MESH]|Peptide Fragments/physiology[MESH]|Peptidyl-Dipeptidase A/*physiology[MESH]|Pneumonia, Viral/etiology/physiopathology/*prevention & control[MESH]|Proto-Oncogene Mas[MESH]|Proto-Oncogene Proteins/physiology[MESH]|Receptor, Angiotensin, Type 1/physiology[MESH]|Receptors, G-Protein-Coupled/physiology[MESH]|Receptors, Virus/*physiology[MESH]
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