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10.1016/j.mehy.2020.109797

http://scihub22266oqcxt.onion/10.1016/j.mehy.2020.109797
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32416411!7252020!32416411
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suck abstract from ncbi

pmid32416411      Med+Hypotheses 2020 ; 142 (ä): 109797
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  • COVID-19 and autism #MMPMID32416411
  • Steinman G
  • Med Hypotheses 2020[Sep]; 142 (ä): 109797 PMID32416411show ga
  • The current pandemic of Covid-19 has created a paradigm for possibly gaining greater insight in two conditions:Studies since the beginning of this century have supported the view that IGF-1 deficiency in the neonate defines the basis of autism. As a result, it appears that interleukin-6 in corona virus-based infections causes reduced defenses because of suppressed IGF-1, especially in older patients. This may also portend an increase of autism in the offspring of gravidas currently affected severely by Covid-19.
  • |Autistic Disorder/*complications[MESH]
  • |Betacoronavirus[MESH]
  • |Biomarkers/metabolism[MESH]
  • |Breast Feeding[MESH]
  • |COVID-19[MESH]
  • |Child, Preschool[MESH]
  • |Coronavirus Infections/*complications[MESH]
  • |Female[MESH]
  • |Humans[MESH]
  • |Infant[MESH]
  • |Infant, Newborn[MESH]
  • |Inflammation[MESH]
  • |Insulin-Like Growth Factor I/deficiency/metabolism[MESH]
  • |Interleukin-6/metabolism[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*complications[MESH]
  • |Pregnancy[MESH]
  • |Pregnancy Complications, Infectious[MESH]


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