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10.1016/j.cmet.2020.04.016 http://scihub22266oqcxt.onion/10.1016/j.cmet.2020.04.016 32402265!7217078!32402265     free     free     free       Cell+Metab 2020 ; 32 (1): 4-5 Nephropedia Template TP {{tp|p=32402265|t=2020. CD4(+) T Cells at the Center of Inflammaging |pdf=|usr=}}{{32402265}} gab.com Text CD4(+) T Cells at the Center of Inflammaging JO: Cell Metab http://www.kidney.de/pget.php?&tw=1&pmid=32402265 #FOAvip Bharath et al., 2020 report that CD4(+) T lymphocytes from aged individuals exhibit defective mitochondrial autophagy, resulting in altered redox metabolism and upregulation of TH17 cytokines, which in turn may contribute to aging-associated chronic inflammation or "inflammaging." Of note, the antiaging drug metformin reverses this autophagy defect and rejuvenates CD4(+) T cell function. Twit Text FOAVip CD4(+) T Cells at the Center of Inflammaging ????Cell Metab http://www.kidney.de/pget.php?&tw=1&pmid=32402265 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32402265 #FOAvip English Wikipedia <ref>{{Cite pmid|32402265}}</ref> CD4(+) T Cells at the Center of Inflammaging #MMPMID32402265 Kroemer G; Zitvogel L Cell Metab 2020[Jul]; 32 (1): 4-5 PMID32402265show ga Bharath et al., 2020 report that CD4(+) T lymphocytes from aged individuals exhibit defective mitochondrial autophagy, resulting in altered redox metabolism and upregulation of TH17 cytokines, which in turn may contribute to aging-associated chronic inflammation or "inflammaging." Of note, the antiaging drug metformin reverses this autophagy defect and rejuvenates CD4(+) T cell function. |*Metformin/pharmacology[MESH] |Aged[MESH] |Aging[MESH] |Autophagy[MESH] |CD4-Positive T-Lymphocytes/metabolism[MESH] |Cytokines/metabolism[MESH] |Humans[MESH] |Inflammation/metabolism[MESH]CD4(+) T Cells at the Center of Inflammaging (epmc).pdf DeepDyve Pubget Overpricing