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10.1038/s41577-020-0331-4

http://scihub22266oqcxt.onion/10.1038/s41577-020-0331-4
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32376901!7201395!32376901
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suck abstract from ncbi


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pmid32376901      Nat+Rev+Immunol 2020 ; 20 (6): 355-362
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  • Pathological inflammation in patients with COVID-19: a key role for monocytes and macrophages #MMPMID32376901
  • Merad M; Martin JC
  • Nat Rev Immunol 2020[Jun]; 20 (6): 355-362 PMID32376901show ga
  • The COVID-19 pandemic caused by infection with SARS-CoV-2 has led to more than 200,000 deaths worldwide. Several studies have now established that the hyperinflammatory response induced by SARS-CoV-2 is a major cause of disease severity and death in infected patients. Macrophages are a population of innate immune cells that sense and respond to microbial threats by producing inflammatory molecules that eliminate pathogens and promote tissue repair. However, a dysregulated macrophage response can be damaging to the host, as is seen in the macrophage activation syndrome induced by severe infections, including in infections with the related virus SARS-CoV. Here we describe the potentially pathological roles of macrophages during SARS-CoV-2 infection and discuss ongoing and prospective therapeutic strategies to modulate macrophage activation in patients with COVID-19.
  • |Animals[MESH]
  • |Betacoronavirus/immunology[MESH]
  • |COVID-19[MESH]
  • |Coronavirus Infections/*complications/*immunology/pathology[MESH]
  • |Humans[MESH]
  • |Inflammation/*etiology/pathology[MESH]
  • |Macrophage Activation/drug effects[MESH]
  • |Macrophages/*immunology[MESH]
  • |Monocytes/immunology[MESH]
  • |Pandemics[MESH]
  • |Pneumonia, Viral/*complications/*immunology/pathology[MESH]


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