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10.1016/j.rmed.2020.105996 http://scihub22266oqcxt.onion/10.1016/j.rmed.2020.105996 32364961!7194970!32364961     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Respir+Med 2020 ; 168 (ä): 105996 Nephropedia Template TP {{tp|p=32364961|t=2020. Severe respiratory SARS-CoV2 infection: Does ACE2 receptor matter?|pdf=|usr=}}{{32364961}} gab.com Text Severe respiratory SARS-CoV2 infection: Does ACE2 receptor matter JO: Respir Med http://www.kidney.de/pget.php?&tw=1&pmid=32364961 #FOAvip SARS-CoV-2 is a novel virus of the Coronaviridiae family that represents a major global health issue. Mechanisms implicated in virus/host cells interaction are central for cell infection and replication that in turn lead to disease onset and local damage. To enter airway and lung epithelia, SARS-CoV-2 attaches to ACE2 receptors by spike (S) glycoproteins. Molecular mechanisms that promote interaction between SARS-CoV-2 virus and host with particular focus on virus cell entry receptor ACE2 are described. We further explore the impact of underlying medical conditions and therapies including renin-angiotensin inhibitors on modulating ACE 2, which is the major SARS-CoV-2 cell entry receptor. Twit Text FOAVip Severe respiratory SARS-CoV2 infection: Does ACE2 receptor matter ????Respir Med http://www.kidney.de/pget.php?&tw=1&pmid=32364961 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32364961 #FOAvip English Wikipedia <ref>{{Cite pmid|32364961}}</ref> Severe respiratory SARS-CoV2 infection: Does ACE2 receptor matter? #MMPMID32364961 Perrotta F; Matera MG; Cazzola M; Bianco A Respir Med 2020[Jul]; 168 (ä): 105996 PMID32364961show ga SARS-CoV-2 is a novel virus of the Coronaviridiae family that represents a major global health issue. Mechanisms implicated in virus/host cells interaction are central for cell infection and replication that in turn lead to disease onset and local damage. To enter airway and lung epithelia, SARS-CoV-2 attaches to ACE2 receptors by spike (S) glycoproteins. Molecular mechanisms that promote interaction between SARS-CoV-2 virus and host with particular focus on virus cell entry receptor ACE2 are described. We further explore the impact of underlying medical conditions and therapies including renin-angiotensin inhibitors on modulating ACE 2, which is the major SARS-CoV-2 cell entry receptor. |*Betacoronavirus/drug effects/physiology[MESH] |*Peptidyl-Dipeptidase A[MESH] |*Receptors, Virus[MESH] |Angiotensin Receptor Antagonists/*pharmacology[MESH] |Angiotensin-Converting Enzyme 2[MESH] |Angiotensin-Converting Enzyme Inhibitors/*pharmacology[MESH] |COVID-19[MESH] |Coronavirus Infections/*virology[MESH] |Host Microbial Interactions/drug effects/physiology[MESH] |Humans[MESH] |Pandemics[MESH] |Pneumonia, Viral/*virology[MESH] |Respiratory Mucosa/metabolism[MESH] |SARS-CoV-2[MESH]Severe respiratory SARS-CoV2 infection: Does ACE2 receptor matter?(epmc).pdf DeepDyve Pubget Overpricing